Results 61 to 70 of about 341,625 (295)

Incidence rates for hemorrhagic stroke, subdural hemorrhage, subarachnoid hemorrhage and traumatic intracranial hemorrhage by drug exposure group.

open access: yes, 2018
Incidence rates for hemorrhagic stroke, subdural hemorrhage, subarachnoid hemorrhage and traumatic intracranial hemorrhage by drug exposure group.
Mattis A. Madsbu (5680358)   +9 more
core   +1 more source

Intracerebral hemorrhages in adults with community associated bacterial meningitis in adults: should we reconsider anticoagulant therapy?

open access: yesPLoS ONE, 2012
ObjectiveTo study the incidence, clinical presentation and outcome of intracranial hemorrhagic complications in adult patients with community associated bacterial meningitis.MethodsNationwide prospective cohort study from all hospitals in the Netherlands,
Barry B Mook-Kanamori   +4 more
doaj   +1 more source

Remote Ischemic Post‐Conditioning Preserves the Neurovascular Unit by Brain‐Derived Gas6 That Drives Monocyte‐Derived Macrophage Reprogramming via Axl and Vimentin after SAH

open access: yesAdvanced Science, EarlyView.
Subarachnoid hemorrhage (SAH) triggers monocyte‐derived macrophage (MDM) infiltration and M1‐like polarization via Vimentin upregulation, leading to neurovascular unit disruption and cognitive dysfunction. Remote ischemic postconditioning (RIPostC) upregulates neuronal Gas6, which activates Axl on MDMs to downregulate Vimentin and drive reparative M2 ...
Yajun Zhu   +14 more
wiley   +1 more source

Coagulation Profile According to Intracranial Hemorrhage Status.

open access: yes, 2014
(−), negative; (+), positive.Coagulation Profile According to Intracranial Hemorrhage Status.
David J. Gemmel (639333)   +5 more
core   +1 more source

Association between Venous Angioarchitectural Features of Sporadic Brain Arteriovenous Malformations and Intracranial Hemorrhage [PDF]

open access: yes, 2015
BACKGROUND AND PURPOSE: Intracranial hemorrhage is the most serious outcome for brain arteriovenous malformations.
H. Kim   +29 more
core   +1 more source

Review and recommendations on management of refractory raised intracranial pressure in aneurysmal subarachnoid hemorrhage

open access: yesVascular Health and Risk Management, 2013
Calvin Hoi Kwan Mak, Yeow Yuen Lu, George Kwok Chu Wong Department of Surgery, Prince of Wales Hospital, Shatin, Hong Kong Abstract: Intracranial hypertension is commonly encountered in poor-grade aneurysmal subarachnoid hemorrhage patients.
Mak CH, Lu YY, Wong GK
doaj  

Inhibition of MEKK3 Prevents Cerebral Cavernous Malformation Progression Via Restoring Endothelial Integrity and Restricting Microglial Activation

open access: yesAdvanced Science, EarlyView.
In this study, we demonstrate that aberrant MEKK3 signaling in endothelial cells and microglia drives the progression of cerebral cavernous malformations (CCMs). Furthermore, we identify DPDH as a potent small‐molecule MEKK3 inhibitor with substantial therapeutic potential for CCM treatment.
Weiwei Zheng   +17 more
wiley   +1 more source

Clinical outcome after first and recurrent hemorrhage in patients with untreated brain arteriovenous malformation [PDF]

open access: yes, 2006
Background and Purpose: The morbidity from spontaneous hemorrhage of untreated brain arteriovenous malformations (AVM) is not well described. Methods: The 241 consecutive AVM patients (mean age 3716 years, 52% women) from the prospective Columbia AVM ...

core  

Intracranial pressure after subarachnoid hemorrhage [PDF]

open access: yes, 2015
Objectives: To describe mean intracranial pressure after aneurysmal subarachnoid hemorrhage, to identify clinical factors associated with increased mean intracranial pressure, and to explore the relationship between mean intracranial pressure and outcome.
T. Zoerle   +6 more
core   +1 more source

Stroke‐Heart Syndrome: Mechanisms, Temporal Phenotypes, and Diagnostic Reasoning

open access: yesAdvanced Science, EarlyView.
Stroke‐heart syndrome describes new or worsened cardiac abnormalities after acute stroke. This Review defines its clinical boundaries, traces autonomic, inflammatory, endothelial, and supply‐demand mechanisms across time, and proposes provisional phenotypes that integrate lesion location, premorbid cardiac substrate, serial biomarkers ...
Fa Lin   +13 more
wiley   +1 more source

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