Results 91 to 100 of about 24,385 (266)

Human preterm infants diagnosed with IUGR have decreased Paneth cell counts compared to preterm non-IUGR control infants.

open access: yes, 2016
Tissues obtained from preterm infants with non-inflammatory surgical small intestinal diseases were stained for presence of goblet and Paneth cells. Infants with a diagnosis of IUGR (n = 9) were compared to age-matched non-IUGR infants (n = 4).
Jörn-Hendrik Weitkamp (218989)   +6 more
core   +1 more source

Narrative Review on Therapies That Influence Inflammatory Responses During Extremely Premature Perinatal Respiratory Transition

open access: yesActa Paediatrica, EarlyView.
ABSTRACT Aims and Methods Advances in neonatal care have extended borderline survival to 22–24 post‐conceptional weeks. Present review discusses approaches for prolonging short pregnancies and prevention of serious morbidities in extremely premature infants born before 28 weeks of pregnancy.
Mikko Hallman
wiley   +1 more source

Catch-up growth occurs in the IUGR offsprings.

open access: yes, 2019
A. Body weights of the controls (CTL) and the IUGR offsprings were recorded at birth (day 0), 2, 7, 14 and 21 days, as well as 2 and 4 months of age. Data are shown as mean ± SD. n = 45; *p .05 vs. CTL. B.
Xinli Wang (300814)   +6 more
core   +1 more source

Quality Care in Midwifery‐Led Birth Centers: Assessing the Disconnect Between Reimbursement and Perinatal Outcomes

open access: yesBirth, EarlyView.
ABSTRACT Background Health care financing is thought to be a driver of health care quality. The purpose of this research was to analyze reimbursement for midwifery‐led US birth centers and to evaluate the association between reimbursement ratios and clinical outcomes.
Sarah Bradbury   +6 more
wiley   +1 more source

Lo IUGR nella nostra popolazione : dati epidemiologici.

open access: yes, 1999
Lo IUGR nella nostra popolazione: dati ...
Balestreri D   +7 more
core  

PP141. The lipid transporters ABCA1 and ABCG1 are differentially expressed in preeclamptic and IUGR placentas

open access: yes, 2012
INTRODUCTION The ATP-binding cassette (ABC) transporter A1 (ABCA1) and ABCG1 are highly expressed in the placenta in various compartments, including the villous syncytiotrophoblast (V-STB) and foetal endothelial cells.
Wenger, F   +5 more
core   +1 more source

From a novel pathogenic SAMD9L variant to cohort‐wide insights: Whole‐genome sequencing highlights somatic genetic rescue and phenotypic heterogeneity

open access: yesBritish Journal of Haematology, EarlyView.
Summary Germline gain‐of‐function variants in sterile alpha motif domain–containing 9‐like (SAMD9L), located on chromosome 7q, cause a multisystem disorder characterized by bone marrow failure, immunodeficiency and variable neurological involvement. Disease evolution is frequently shaped by somatic genetic rescue (SGR), most commonly through monosomy 7,
Hadjer Dellal   +10 more
wiley   +1 more source

Increased ubiquitination and reduced plasma membrane trafficking of placental amino acid transporter SNAT-2 in human IUGR

open access: yesClinical science, 2015
Inhibition of placental mechanistic target of rapamycin (mTOR) signalling, which activates NEDD4-2 (neural precursor cell expressed developmentally down-regulated protein 4-2) ubiquitin ligase leading to increased sodium-coupled neutral amino acid ...
Yi-Yung Chen   +5 more
semanticscholar   +1 more source

Clinical characteristics of IUGR cases.

open access: yes, 2018
Clinical characteristics of IUGR cases.
Gina Capodanno (5029049)   +11 more
core   +1 more source

USP34 Haploinsufficiency as a Cause of Neurodevelopmental Phenotypes

open access: yesClinical Genetics, EarlyView.
Heterozygous loss‐of‐function variants in USP34 cause a novel neurodevelopmental disorder characterized by global developmental delay, speech impairment, autism, hypotonia, craniofacial dysmorphism, and distal limb anomalies. Disrupted Wnt/β‐catenin signaling via reduced Axin stabilization refines gene‐specific contributions within 2p15p16.1 ...
Helena Wigoda   +10 more
wiley   +1 more source

Home - About - Disclaimer - Privacy