Japanese encephalitis virus NS1 inhibits IFN-β production by interacting with DDX3X. [PDF]
Dong H +5 more
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Vector competence of European <i>Aedes</i> mosquito species for Japanese encephalitis virus under fluctuating temperature conditions. [PDF]
Ciećkiewicz AM +8 more
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Sheep serve as amplifying hosts of Japanese encephalitis virus, increasing the risk of human infection. [PDF]
Zhang H +12 more
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Rates of infection with Japanese encephalitis virus of two culicine species of mosquito in Taiwan.
Takeshi Okuno +4 more
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Zinc finger protein ZNF33B facilitates Japanese encephalitis virus replication by regulating m<sup>6</sup>A modification on Ifih1 and Irf3 transcripts to antagonize antiviral response. [PDF]
Du J +7 more
europepmc +1 more source
Alterations in iron levels in Japanese encephalitis virus infection.
A Mathur +2 more
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Managing Japanese Encephalitis Virus as a Veterinary Infectious Disease Through Animal Surveillance and One Health Control Strategies. [PDF]
Park JY, Lee HM.
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Pathobiology of Japanese encephalitis virus infection
Molecular Aspects of Medicine, 2021Japanese encephalitis virus (JEV) is a flavivirus, spread by the bite of carrier Culex mosquitoes. The subsequent disease caused is Japanese encephalitis (JE), which is the leading global cause of virus-induced encephalitis. The disease is predominant in the entire Asia-Pacific region with the potential of global spread.
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Japanese encephalitis virus glycoproteins
Virology, 1973Abstract Mature Japanese encephalitis (JE) virus, or N-form virus, contained three structural proteins: V-1, V-2, and V-3. The large membrane protein V-3 was glycosylated, whereas both V-1 (the small membrane protein) and V-2 (the nucleocapsid protein) were not. Intracellular (I-form), immature virions from infected chick embryo cells did not contain
D, Shapiro, K A, Kos, P K, Russel
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