Results 201 to 210 of about 976,686 (361)
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira +14 more
wiley +1 more source
Comparison of Standard Setting Procedures to Establish Defensible Passing Standards for Clinical Skills Assessment: Angoff, Borderline Group, Contrasting Groups and Patient Safety Methods. [PDF]
Tappan RS +4 more
europepmc +1 more source
Judging religion ? : law, politics and freedom facing blasphemy in democracy
Anastasia Colosimo
openalex +1 more source
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian +10 more
wiley +1 more source
Construction and validity of an instrument for assessing the competencies of resident nurses in neonatology. [PDF]
Araujo MM +3 more
europepmc +1 more source
LG Uplus System with Multi-Speaker IDs and Discriminator-based Sub-Judges for the WildSpoof Challenge [PDF]
Park, Jinyoung, Jang, Won, Park, Jiwoong
openalex
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath +13 more
wiley +1 more source
CPJ: Explainable Agricultural Pest Diagnosis via Caption-Prompt-Judge with LLM-Judged Refinement [PDF]
Wentao Zhang +4 more
openalex
YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
wiley +1 more source

