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Combination Therapy for Dendritic Keratitis with Acyclovir and Vidarabine
Journal of Ocular Pharmacology and Therapeutics, 1987We treated 32 patients with dendritic keratitis with a combination of acyclovir 3% ointment and vidarabine 3% ointment or acyclovir and placebo. Patients with acyclovir alone healed in an average time of 7.7 days, while patients on the combination healed in an average of 6 days.
J, Colin +3 more
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[Zona ophthalmica and dendritic keratitis].
Canadian journal of ophthalmology. Journal canadien d'ophtalmologie, 1976Two cases of herpes zoster ophthalmicus with dendritic keratitis are reported. Virological studies confirmed the double infection with herpes simplex type 1 virus in the corneal lesions and herpes zoster virus in the cutaneous lesions. We suggest the use of the immunoperoxidase test to identify the viral agent mainly because of its rapid and specific ...
M Y, Laflamme +3 more
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Journal of Pediatric Ophthalmology & Strabismus, 1994
J R, Bierly, H B, Ostler
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J R, Bierly, H B, Ostler
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CRYOTHERAPY FOR DENDRITIC KERATITIS
Medical Journal of Australia, 1969E J, Milverton, A B, Hunyor
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TREATMENT OF DENDRITIC KERATITIS WITH QUININE BISULFATE
Archives of Ophthalmology, 1937Dendritic keratitis seems to be a much more uncommon disease in the Middle West than in the New England states. Gundersen 1 reported on the treatment of two hundred and twenty-one patients with dendritic keratitis seen within forty-two months.
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Acanthamoeba keratitis presenting as dendritic keratitis in a soft contact lens wearer.
Chang Gung medical journal, 2002Acanthamoeba keratitis is a rare cause of corneal infection in Taiwan, which can result in devastating visual outcomes. A 37-year-old woman, who wore soft contact lenses, suffered from severe pain in her left eye. Biomicroscopy revealed dendritic keratitis, radial keratoneuritis, and fine keratic precipitates on her cornea.
Edward Yip, Yeung +2 more
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[Dendritic keratitis following corneal foreign body].
Klinische Monatsblatter fur Augenheilkunde, 1986Traumatogenic herpetic eye disease may be initiated via several pathophysiologic pathways. Trauma of the trigeminal ganglion or adequate systemic alterations are the most effective means of destabilizing ganglionic latency of the herpesvirus with consequent neuronal peripheral virus shedding.
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