Results 201 to 210 of about 273,215 (316)

RECURRENCE OF HENOCH-SCHÖNLEIN PURPURA GLOMERULONEPHRITIS IN TRANSPLANTED KIDNEY,S1,2

open access: bronze, 1974
Tadla Baliah   +5 more
openalex   +1 more source

Genotype–Phenotype Correlation in TTC7A‐Associated Gastrointestinal Defects and Immunodeficiency Syndrome 1

open access: yesAmerican Journal of Medical Genetics Part A, EarlyView.
ABSTRACT Gastrointestinal defects and immunodeficiency syndrome 1 (GIDID1) is a rare autosomal recessive disorder caused by biallelic variants in TTC7A. GIDID1 is characterized by a broad clinical spectrum ranging from very early‐onset inflammatory bowel disease (VEOIBD) to multiple intestinal atresia (MIA) with or without immunological manifestations.
Julia Imhoff   +8 more
wiley   +1 more source

WCN24-388 A STEP TOWARDS BRIDGING THE GENDER GAP IN DECEASED DONOR KIDNEY TRANSPLANTATION

open access: yesKidney International Reports
Khushboo Saxena   +14 more
doaj   +1 more source

Opportunities and challenges with the implementation of normothermic machine perfusion in kidney transplantation. [PDF]

open access: yesNat Commun
Rabelink TJ   +6 more
europepmc   +1 more source

TRANSPLANTATION OF MALIGNANT MELANOMA WITH CADAVER KIDNEY

open access: bronze, 1972
D. Jeremy   +4 more
openalex   +1 more source

The Vietnamese swine as a translational model of invasive ductal carcinoma of the breast

open access: yesAnimal Models and Experimental Medicine, EarlyView.
The flow diagram outlines the chemical induction of breast tumors through a single intraperitoneal dose of MNU. Tumor development was assessed using ultrasound and clinical examination. After 14 weeks, mammary tissue was collected and histologically analyzed to evaluate structural similarities between sows and humans, the carcinogenic potential of MNU,
Claudia Elizabeth Vera‐Tizatl   +6 more
wiley   +1 more source

Construction of pathogenic Sec16a mutation mouse model using CRISPR/Cas9

open access: yesAnimal Models and Experimental Medicine, EarlyView.
Yaqiang Hu et al. engineered a pathogenic Sec16a mutant mouse model using CRISPR/Cas9 technology. They observed that the Sec16a mutant mice displayed diminished learning and memory capabilities, along with a limb‐clasping phenotype upon tail suspension.
Yaqiang Hu   +6 more
wiley   +1 more source

Home - About - Disclaimer - Privacy