Results 61 to 70 of about 2,057,922 (254)

Transesophageal echocardiography measurement of left atrial appendage size.

open access: yes, 2022
Left atrial appendage ostial diameter and depth were measured at 0°, 45°, 90°, and 135°.
Yoichi Takaya (14197311)   +6 more
core   +1 more source

A Spatiotemporal Single‐Cell Atlas Uncovers Dysregulated ECM Dynamics and Septal Remodeling Arrest in Human Ventricular Septal Defects

open access: yesAdvanced Science, EarlyView.
Integrating single‐cell and spatial transcriptomics reveals human ventricular septal defect (VSD) as a disease of microenvironmental insufficiency and structural remodeling arrest. Attenuated endothelial‐fibroblast crosstalk, alongside downregulated THBS1/TGF‐β1 and MMP2 signaling networks, impairs the endothelial‐to‐mesenchymal transition (EndoMT) and
Xiaoyuan Zhang   +9 more
wiley   +1 more source

Left Atrial Appendage Occlusion Guided Only by Transesophageal Echocardiography

open access: yesCardiology Research and Practice, 2019
Aims. To investigate a new method of left atrial appendage occlusion without fluoroscopy. Methods and Results. We performed left atrial appendage occlusion for 14 patients with atrial fibrillation in our hospital. All of the surgeries were completed in a
Jinlong Zhao   +9 more
doaj   +1 more source

Percutaneous management of left atrial appendage perforation during device closure. [PDF]

open access: yes, 2014
A 76-year-old male patient was admitted for percutaneous left atrial appendage (LAA) closure because of chronic atrial fibrillation and a history of gastrointestinal bleeding under oral anticoagulation. The procedure was complicated by perforation of the
Bernhard Meier   +8 more
core   +1 more source

Ischemic Postconditioning Protects Aged Myocardium Against Ischemia/Reperfusion Injury by Regulating Mitochondrial Dynamics via piR‐4913

open access: yesAdvanced Science, EarlyView.
A tiny RNA tips the scales of cardiac injury in aging. piR‐4913 recruits OGT to Drp1 and drives its O‐GlcNAcylation at Thr604, promoting excessive mitochondrial fission that aggravates ischemia‐reperfusion injury. Ischemic postconditioning silences this pathway to restore mitochondrial balance and protect the aged myocardium, revealing piR‐4913 as a ...
Xiangyu Zhao   +11 more
wiley   +1 more source

Device-Assisted Left Atrial Appendage Exclusion: From Basic Sciences to Clinical Applications

open access: yesJournal of Cardiovascular Development and Disease
Device-assisted left atrial appendage exclusion plays a crucial role in the prevention of fatal ischemic complications in patients with atrial fibrillation and contraindications to anticoagulation treatment. Various devices with different safety profiles
Julia Izabela Karpierz   +5 more
doaj   +1 more source

Two‐Year Outcomes With a Next‐Generation Left Atrial Appendage Device: Final Results of the PINNACLE FLX Trial

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, 2023
Background The PINNACLE FLX (Protection Against Embolism for Non‐valvular AF [Atrial Fibrillation] Patients: Investigational Device Evaluation of the Watchman FLX LAA [Left Atrial Appendage] Closure Technology) trial evaluated the safety and efficacy of ...
Shephal K. Doshi   +18 more
doaj   +1 more source

Left atrial appendage: anatomy and pulsed Doppler flow profile.

open access: yes, 2015
LA: left atrium, LAA: left atrium appendage. Values are mean±SD.Left atrial appendage: anatomy and pulsed Doppler flow profile.
Elena Nobili (314243)   +6 more
core   +1 more source

Left Atrial Appendage Exclusion for Atrial Fibrillation [PDF]

open access: yesHeart Failure Clinics, 2014
Percutaneous left atrial appendage (LAA) closure is being increasingly used as a treatment strategy to prevent stroke in patients with atrial fibrillation (AF) who have contraindications to anticoagulants. Several approaches and devices have been developed in the last few years, each with their own unique set of advantages and disadvantages.
Faisal F, Syed   +3 more
openaire   +3 more sources

Stroke‐Heart Syndrome: Mechanisms, Temporal Phenotypes, and Diagnostic Reasoning

open access: yesAdvanced Science, EarlyView.
Stroke‐heart syndrome describes new or worsened cardiac abnormalities after acute stroke. This Review defines its clinical boundaries, traces autonomic, inflammatory, endothelial, and supply‐demand mechanisms across time, and proposes provisional phenotypes that integrate lesion location, premorbid cardiac substrate, serial biomarkers ...
Fa Lin   +13 more
wiley   +1 more source

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