Results 301 to 310 of about 895,463 (333)
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Hematology/Oncology Clinics of North America, 2008
Acute myeloid leukemia (AML) is a heterogeneous group of leukemias that result from clonal transformation of hematopoietic precursors through the acquisition of chromosomal rearrangements and multiple gene mutations. As a result of highly collaborative clinical research by pediatric cooperative cancer groups worldwide, disease-free survival has ...
Jeffrey E, Rubnitz +2 more
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Acute myeloid leukemia (AML) is a heterogeneous group of leukemias that result from clonal transformation of hematopoietic precursors through the acquisition of chromosomal rearrangements and multiple gene mutations. As a result of highly collaborative clinical research by pediatric cooperative cancer groups worldwide, disease-free survival has ...
Jeffrey E, Rubnitz +2 more
+7 more sources
Chronic myeloid leukemia: 2022 update on diagnosis, therapy, and monitoring
American journal of hematology/oncology, 2022Chronic Myeloid Leukemia (CML) is a myeloproliferative neoplasm with an incidence of 1–2 cases per 100 000 adults. It accounts for approximately 15% of newly diagnosed cases of leukemia in adults.
E. Jabbour, H. Kantarjian
semanticscholar +1 more source
Hematology/Oncology Clinics of North America, 2009
The evolution of acute myeloid leukemia (AML) classification reflects greater understanding of the AML pathogenesis. The 2008 World Health Organization classification incorporated cytogenetic and molecular genetic findings and introduced important prognostic correlations.
Amy, Heerema-McKenney, Daniel A, Arber
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The evolution of acute myeloid leukemia (AML) classification reflects greater understanding of the AML pathogenesis. The 2008 World Health Organization classification incorporated cytogenetic and molecular genetic findings and introduced important prognostic correlations.
Amy, Heerema-McKenney, Daniel A, Arber
openaire +2 more sources
Current Opinion in Hematology, 1997
Progress has been made in understanding BCR-ABL-positive leukemias. A new transcript (p230BCR-ABL) has been characterized that is associated with Ph-positive chronic neutrophilic leukemia. The ATM protein appears to be a regulator of ABL activity in response to irradiation damage.
John M. Goldman, Michael W. Deininger
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Progress has been made in understanding BCR-ABL-positive leukemias. A new transcript (p230BCR-ABL) has been characterized that is associated with Ph-positive chronic neutrophilic leukemia. The ATM protein appears to be a regulator of ABL activity in response to irradiation damage.
John M. Goldman, Michael W. Deininger
openaire +4 more sources
Chronic Myeloid Leukemia [PDF]
New England Journal of Medicine, 1999 Over the past twenty years, clinical and laboratory studies have led to important new insights into the biology of chronic myeloid leukemia. Basic science has defined the molecular pathogenesis of chronic myeloid leukemia (CML) as unregulated signal transduction by the Bcr-Abl tyrosine kinase.
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Annals of the New York Academy of Sciences, 1964
The incidence of myeloid leukemia is elevated in RF mice acutely exposed early in adult life to 50 to 400 rads of whole-body x radiation. The relation between incidence and dose is complex, the yield of leukemias declining as the dose is increased above an optimal level.
Arthur C. Upton +2 more
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The incidence of myeloid leukemia is elevated in RF mice acutely exposed early in adult life to 50 to 400 rads of whole-body x radiation. The relation between incidence and dose is complex, the yield of leukemias declining as the dose is increased above an optimal level.
Arthur C. Upton +2 more
openaire +3 more sources
Current Opinion in Oncology, 1992
Chronic myeloid leukemia is a clonal myeloproliferative disorder of a pluripotent stem cell with a specific cytogenetic abnormality, the Philadelphia chromosome, involving myeloid, erythroid, megakaryocytic, B lymphoid, and sometimes T lymphoid cells but not marrow fibroblasts.
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Chronic myeloid leukemia is a clonal myeloproliferative disorder of a pluripotent stem cell with a specific cytogenetic abnormality, the Philadelphia chromosome, involving myeloid, erythroid, megakaryocytic, B lymphoid, and sometimes T lymphoid cells but not marrow fibroblasts.
openaire +5 more sources