Results 181 to 190 of about 4,515 (208)
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Journal of Skin and Sexually Transmitted Diseases
Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory dermatosis, typically characterized by ivory-white, atrophic plaques, affecting the anogenital region most commonly.
Geethanjali Sahadevan
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Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory dermatosis, typically characterized by ivory-white, atrophic plaques, affecting the anogenital region most commonly.
Geethanjali Sahadevan
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Journal of the European Academy of Dermatology and Venereology, 2021
Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory sclerotic disease that primarily involves the genital mucosa. Isolated extra-genital involvement is extremely rare, accounting only for 2.5% of cases.
Ozlem Ozbagcivan +2 more
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Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory sclerotic disease that primarily involves the genital mucosa. Isolated extra-genital involvement is extremely rare, accounting only for 2.5% of cases.
Ozlem Ozbagcivan +2 more
semanticscholar +1 more source
Photosensitive generalised guttate lichen sclerosus et atrophicus: A rare presentation
Australasian Journal of Dermatology, 20211. Ackerman AB, Viragh PA, Chongchitnant N. Neoplasms with Follicular Differentiation. Philadelphia, PA: Lea & Febiger, 1993; 552. 2. Markowitz O, Schwartz M, Feldman E et al.
B. Behera +4 more
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Acrosyringeal variant of extragenital lichen sclerosus et atrophicus
Journal of cutaneous pathology, 2020Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory dermatosis of the anogenital area, and approximately 15% to 20% of patients with LSA have extragenital lesions.
Seon Bok Lee +6 more
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Journal of the European Academy of Dermatology and Venereology, 2020
The Koebner phenomenon refers to triggering of a skin disorder at sites of cutaneous trauma or any other dermatosis.1 We report an unusual colocalization of pigmentary mosaicism and extragenital lichen sclerosus et atrophicus (EG-LSA) in a child.
P. Sharma, S. Sonthalia, R. Happle
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The Koebner phenomenon refers to triggering of a skin disorder at sites of cutaneous trauma or any other dermatosis.1 We report an unusual colocalization of pigmentary mosaicism and extragenital lichen sclerosus et atrophicus (EG-LSA) in a child.
P. Sharma, S. Sonthalia, R. Happle
semanticscholar +1 more source
Lichen Sclerosus et Atrophicus
2016Lichen sclerosus et atrophicus (LSA) is a chronic inflammatory dermatosis that has a predilection for the anogenital region. Vulval and perianal lesions present as white papules or plaques. There may be associated pallor, atrophy, hyperkeratosis, or scarring. Extragenital lesions involve the upper trunk, axillae, buttocks, and lateral thighs.
Hee Jin Kim, Robert A. Schwartz
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Lichen sclerosus (et atrophicus)
2017Lichen sclerosus ist eine lymphozytar vermittelte, chronisch entzundliche Hauterkrankung. Es werden Definition, Diagnostik und Therapie besprochen.
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LICHEN SCLEROSUS ET ATROPHICUS
Archives of Dermatology, 1940It is our belief that lichen sclerosus et atrophicus, which has often been designated "lichen planus sclerosus et atrophicus" (Hallopeau 1 ), "lichen planus morphoeicus" (Crocker 2 ) or "white spot disease" (Johnston and Sherwell 3 ) in the German literature, is a distinct entity and can be distinguished both clinically and pathologically from atrophic
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Lichen Sclerosus et Atrophicus
Archives of Dermatology, 1961Although in the past lichen sclerosus et atrophicus (LSA) has been considered to be related to several other dermatoses,10most authors6,9,27,34,36now accept the opinion of Kyrle14that it is a separate entity. Nevertheless, the description of the histologic structure of LSA found in textbooks is far from uniform.
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[Lichen sclerosus et atrophicus].
Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete, 1991A review of lichen sclerosus et atrophicus is presented in this article. Clinical manifestations, possible complications and histology are described. Particular emphasis is given to the aetiological hypotheses, which include genetic, autoimmune, hormonal and infectious mechanisms. Finally, the various treatments are discussed.
R, Feldmann, M, Harms
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