Results 91 to 100 of about 4,214 (224)

Thermoregulatory and plasma neurobiomarker responses to heat tolerance assessment in exertional heat illness and matched controls

open access: yesExperimental Physiology, EarlyView.
Abstract Exercise in the heat and elevated tissue temperature are associated with neuronal stress and changes in blood–brain barrier (BBB) function. More severe thermal insult may manifest as exertional heat illness (EHI) and complication by central nervous system (CNS) dysfunction may persist post‐EHI.
Tom W. Palin   +15 more
wiley   +1 more source

[Descent gradient of maximal expiratory flow-volume curve and uneven distribution of ventilation].

open access: yesNihon Kyobu Shikkan Gakkai zasshi, 1985
Uzawa, Takeshi   +4 more
openaire   +2 more sources

Impact of living and training at low altitude for several years on endurance performance at higher altitude: Myth or reality

open access: yesExperimental Physiology, EarlyView.
Abstract The physiological adaptations of long‐term high‐altitude residents without high‐altitude genetic ancestry remain poorly characterized. This study investigated whether permanent residence at low altitude (1200 m) influences performance at higher altitude (2500 m). We compared exercise responses at 2500 m in endurance‐trained athletes living for
Fabienne Durand   +2 more
wiley   +1 more source

Age‐related loss of hysteresis in the spontaneous vascular sympathetic baroreflex is associated with altered neural rather than mechanical control

open access: yesExperimental Physiology, EarlyView.
Abstract The vascular sympathetic baroreflex is critical for stabilising blood pressure. A notable feature of this reflex is hysteresis, a directional asymmetry in responsiveness during rising versus falling pressure that is evident in young adults but attenuated in older age.
Guto Wyn Hughes   +5 more
wiley   +1 more source

Impact of chronic obstructive pulmonary disease on the prognosis of patients with extensive-stage small-cell lung cancer treated with chemoimmunotherapy. [PDF]

open access: yesTransl Lung Cancer Res
Jiang Y   +13 more
europepmc   +1 more source

The mitochondrial‐targeted antioxidant SkQ1 prevents skeletal muscle mitochondrial‐apoptotic but not necroptotic signalling during ovarian cancer

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend An evaluation of the degree to which mitochondrial hydrogen peroxide emission (mH2O2)‐mediated apoptotic and necroptotic signalling contributes to skeletal muscle atrophy in an orthotopic epithelial ovarian cancer (EOC) model. To determine whether attenuating mH2O2 could prevent regulated cell death signalling and mitigate muscle
Shahrzad Khajehzadehshoushtar   +15 more
wiley   +1 more source

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