Results 81 to 90 of about 652,444 (293)

The embedding of a cyclic permutable subgroup in a finite group. II [PDF]

open access: yes, 2004
In two previous papers we established the structure of the normal closure of a cyclic permutable subgroup $A$ of a finite group, first when $A$ has odd order and second when $A$ has even order, but with an extra hypothesis that was unnecessary in the odd
Cossey, Peter (John)   +3 more
core   +1 more source

Epigenetic silencing of the liver‐specific lncRNA LUNAR promotes liver cancer progression via NOTCH activation

open access: yesMolecular Oncology, EarlyView.
LUNAR is a liver‐specific long noncoding RNA (lncRNA) that is highly expressed in normal liver but becomes epigenetically silenced in hepatocellular carcinoma through promoter hypermethylation. Loss of LUNAR is associated with NOTCH activation, epithelial–mesenchymal transition, and metastasis, whereas restoring LUNAR restrains metastatic progression ...
Se Ha Jang   +9 more
wiley   +1 more source

Theoretical Researches about u-Maximal Subgroups and Its Applications in Charactering IntuG

open access: yesJournal of Chemistry, 2019
Let G be a finite group and u be the class of all finite supersoluble groups. A supersoluble subgroup U of G is called u-maximal in G if for any supersoluble subgroup V of G containing U, V=U.
Li Zhang, Zheng-Qun Cai
doaj   +1 more source

Nilpotent Fuzzy Subgroups

open access: yesMathematics, 2018
In this paper, we introduce a new definition for nilpotent fuzzy subgroups, which is called the good nilpotent fuzzy subgroup or briefly g-nilpotent fuzzy subgroup.
Elaheh Mohammadzadeh, Rajab Ali Borzooei
doaj   +1 more source

Single‐cell DNA methylation profiling: Technologies, computation, and applications in precision oncology

open access: yesMolecular Oncology, EarlyView.
Single‐cell DNA methylation (scDNAme) profiling maps epimutational clonal evolution, revealing mechanisms of malignancy and therapeutic resistance across diverse cancer types. By providing a high‐resolution landscape of intratumoral heterogeneity, these technologies empower precise patient stratification, guide the development of enhanced ...
Ik Soo Kim
wiley   +1 more source

Fischer-Clifford Matrices and Character Table of the Maximal Subgroup (29:(L3(4)):2 of U6(2):2

open access: yesInternational Journal of Mathematics and Mathematical Sciences, 2019
The automorphism group U6(2):2 of the unitary group U6(2)≅Fi21 has a maximal subgroup G¯ of the form (29:(L3(4)):2 of order 20643840. In this paper, Fischer-Clifford theory is applied to the split extension group G¯ to construct its character table. Also,
Abraham Love Prins   +1 more
doaj   +1 more source

On endomorphisms of groups of order 32 with maximal subgroups C4 x C2 x C2; pp. 105–120 [PDF]

open access: yesProceedings of the Estonian Academy of Sciences, 2014
It is proved that each group of order 32, which has a maximal subgroup isomorphic to C4 x C2 x C2, is determined by its endomorphism semigroup in the class of all groups.
Piret Puusemp, Peeter Puusemp
doaj   +1 more source

Maximal Sylow Subgroups of a Given Group [PDF]

open access: yesProceedings of the National Academy of Sciences, 1942
Not ...
openaire   +3 more sources

Profiling neoadjuvant therapy response in rectal cancer using meta‐analysis of publicly available transcriptomic RNA‐seq datasets

open access: yesMolecular Oncology, EarlyView.
This study integrates publicly available transcriptomic datasets to identify molecular signatures associated with response to neoadjuvant chemoradiotherapy in locally advanced rectal cancer. By analyzing a combination of multiple cohorts with bioinformatics approaches, we reveal biological pathways and immune‐related features that may improve ...
Aleksandra Stanojevic   +10 more
wiley   +1 more source

Aquaporin‐3 and aquaporin‐5 impact the development of pancreatic ductal adenocarcinoma spheroids

open access: yesFEBS Open Bio, EarlyView.
Schematic representation of the role of aquaporin‐3 (AQP3) and aquaporin‐5 (AQP5) in pancreatic ductal adenocarcinoma (PDAC). Both proteins are upregulated in PDAC and are associated with tumor progression and metastatic potential. Silencing AQP3 or AQP5 in PDAC spheroids results in decreased diameter, area, and overall growth, underscoring their key ...
Catarina Pimpão   +3 more
wiley   +1 more source

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