Results 91 to 100 of about 178,986 (267)

Incident study of medication errors in drug use processes: prescription, transcription, validation, preparation, dispensing, and administering in the hospital environment [PDF]

open access: green, 2009
Lourdes Pastó-Cardona   +8 more
openalex   +1 more source

Medication errors: They continue

open access: yesJournal of Anaesthesiology Clinical Pharmacology, 2019
Rashmi Salhotra, Asha Tyagi
doaj   +1 more source

Prevalence of medication errors and its related factors in Iranian nurses: an updated systematic review and meta-analysis

open access: yesBMC Nursing
Background Nurses may make medication errors during the implementation of therapeutic interventions, which initially threaten the patient’s health and safety and prolong their hospital stay.
Hadis Fathizadeh   +4 more
doaj   +1 more source

Glycosylated LGALS3BP is highly secreted by bladder cancer cells and represents a novel urinary disease biomarker

open access: yesMolecular Oncology, EarlyView.
Urinary LGALS3BP is elevated in bladder cancer patients compared to healthy controls as detected by the 1959 antibody–based ELISA. The antibody shows enhanced reactivity to the high‐mannose glycosylated variant secreted by cancer cells treated with kifunensine (KIF).
Asia Pece   +18 more
wiley   +1 more source

Medication Errors in Pediatrics

open access: yesPediatrics and Neonatology, 2013
Chih-Jung Chen
doaj   +1 more source

Evaluation of the frequency and type of medication prescribing errors in the NICU of hospitals in Yazd

open access: yesحیات, 2016
Background & Aim: Medication errors are one of the most common medical errors and these errors have a double importance in neonatal intensive care unit.
Naiire Salmani, Shirin Hasanvand
doaj  

Survivin and Aurora Kinase A control cell fate decisions during mitosis

open access: yesMolecular Oncology, EarlyView.
Aurora A interacts with survivin during mitosis and regulates its centromeric role. Loss of Aurora A activity mislocalises survivin, the CPC and BubR1, leading to disruption of the spindle checkpoint and triggering premature mitotic exit, which we refer to as ‘mitotic slippage’.
Hana Abdelkabir   +2 more
wiley   +1 more source

CDK11 inhibition induces cytoplasmic p21WAF1 splice variant by p53 stabilisation and SF3B1 inactivation

open access: yesMolecular Oncology, EarlyView.
CDK11 inhibition stabilises the tumour suppressor p53 and triggers the production of an alternative p21WAF1 splice variant p21L, through the inactivation of the spliceosomal protein SF3B1. Unlike the canonical p21WAF1 protein, p21L is localised in the cytoplasm and has reduced cell cycle‐blocking activity.
Radovan Krejcir   +12 more
wiley   +1 more source

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