Results 251 to 260 of about 1,652,787 (364)

The Burden of Adverse Drug Reactions in Africa in the Context of Pharmacogenetics-Based Clinical Guidelines. [PDF]

Clin Pharmacol Ther
Scholefield J, Mazhindu TA, Nagy M, Twesigomwe D, Agesa G, Masimirembwa C.   +5 more
europepmc   +1 more source

International meeting: new diagnostic tests are urgently needed to treat patients with Chaga's disease [PDF]

, 2007
Campaign for Access to Essential Medicines, Medecins Sans Frontieres   +1 more
core   +1 more source

Holism of Body and Mind in Hippocratic Medicine and Greek Tragedy

, 2020
Elizabeth M. Craik
openalex   +1 more source

New York academy of medicine: Section on laryngology and rhinology [PDF]

, 1910
Harmon Smith
openalex   +1 more source

Network divergence analysis identifies adaptive gene modules and two orthogonal vulnerability axes in pancreatic cancer

Molecular Oncology, EarlyView.
Tumors contain diverse cellular states whose behavior is shaped by context‐dependent gene coordination. By comparing gene–gene relationships across biological contexts, we identify adaptive transcriptional modules that reorganize into distinct vulnerability axes.
Brian Nelson, Lyanne Delgado‐Coka, Natalia Marchenko, Luisa F. Escobar‐Hoyos, Kenneth R. Shroyer, Alisa Yurovsky, Trey Ideker, Gábor Balázsi, Thomas MacCarthy, Scott Powers   +9 more
wiley   +1 more source

Policy barriers to drug repurposing in Europe: different stakeholder perspectives identified during survey-based shortlisting of key challenges. [PDF]

Health Policy Open
Gyöngyösi K, Petykó ZI, Hosszú D, Pantziarka P, van der Meer HG, Lo DC, Csanádi M, Obeng GD, Kaló Z, Inotai A.   +9 more
europepmc   +1 more source

RIPK4 function interferes with melanoma cell adhesion and metastasis

Molecular Oncology, EarlyView.
RIPK4 promotes melanoma growth and spread. RIPK4 levels increase as skin lesions progress to melanoma. CRISPR/Cas9‐mediated deletion of RIPK4 causes melanoma cells to form less compact spheroids, reduces their migratory and invasive abilities and limits tumour growth and dissemination in mouse models.
Norbert Wronski, Sławomir Lasota, Ewelina Madej, Anna A. Brożyna, Małgorzata Szczygieł, Agnieszka Harazin‐Lechowska, Jan Czerbniak, Janusz Rys, Jaroslaw Czyz, Agnieszka Wolnicka‐Glubisz   +9 more
wiley   +1 more source

Complementary Medicine Use and Perceptions of It in Victoria, Australia: A Statewide Cross-Sectional Survey. [PDF]

Nutrients
Naseri K, Shahin W, Allahham A, Bilal H, de Courten B, Thrimawithana TR.   +5 more
europepmc   +1 more source

Fringe Medicine

BMJ, 1960
openaire   +2 more sources

COMP–PMEPA1 axis promotes epithelial‐to‐mesenchymal transition in breast cancer cells

Molecular Oncology, EarlyView.
This study reveals that cartilage oligomeric matrix protein (COMP) promotes epithelial‐to‐mesenchymal transition (EMT) in breast cancer. We identify PMEPA1 (protein TMEPAI) as a novel COMP‐binding partner that mediates EMT via binding to the TSP domains of COMP, establishing the COMP–PMEPA1 axis as a key EMT driver in breast cancer.
Konstantinos S. Papadakos, Gilar Gorji‐bahri, Lejla Gradjan, Julia Zajac, Kacper Gil, Yvonne Thokozile Nduku, Anna M. Blom   +6 more
wiley   +1 more source