Results 201 to 210 of about 13,793 (230)
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Minimal Hepatic Encephalopathy
Clinics in Liver DiseaseMinimal hepatic encephalopathy (MHE) is a pervasive frequent complication of cirrhosis of any etiology. The diagnosis of MHE is difficult as the standard neurologic examination is essentially within normal limits. None of the symptoms and signs of overt HE is present in a patient with MHE, such as confusion, disorientation, or asterixis.
Rachel, Redfield +2 more
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Attention Deficits in Minimal Hepatic Encephalopathy
Metabolic Brain Disease, 2001Minimal hepatic encephalopathy (HE) is characterized by a decrease of psychomotor speed, and deficits in visual perception, visuo-spatial orientation, and visuo-constructive abilities. Attention deficits have also been proposed to be part of the syndrome. Several attempts were made in the past to elaborate suitable psychometric means for the assessment
K, Weissenborn +4 more
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Treatment of minimal hepatic encephalopathy
Hepatology Research, 2008Several reports, conducted as a placebo‐controlled, double‐blind study, presented a favorable result in treating MHE. They included branched chain aminoacid, fluzazenil‐a benzodiazepin receptor antagonist, lactulose, lactitol, and L‐ornithin‐L‐aspartate. Lactulose and lactitol have been shown to be effective in MHE.
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Rifaximin Therapy in Minimal Hepatic Encephalopathy Cirrhotics
American Journal of Gastroenterology, 2011To the Editor: We read the interesting trial by Sidhu et al. (1) dealing with the efficacy of rifaximin in improving health-related quality of life (HRQOL) in minimal hepatic encephalopathy (MHE) cirrhotics as compared with placebo. The authors concluded that rifaximin is significantly associated with an improvement of HRQOL, as assessed by sickness ...
Zullo, A, Hassan, Cesare, Lorenzetti, R.
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Probiotic Yogurt for the Treatment of Minimal Hepatic Encephalopathy
The American Journal of Gastroenterology, 2008Minimal hepatic encephalopathy (MHE), the preclinical stage of overt hepatic encephalopathy (OHE), is a significant condition affecting up to 60% of cirrhotics. All MHE therapies modify gut microflora, but consensus regarding MHE treatment and long-term adherence studies is lacking.
Jasmohan S, Bajaj +9 more
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Lactulose Management of Minimal Hepatic Encephalopathy
Gastroenterology Nursing, 2019Minimal hepatic encephalopathy has been shown to increase risk of involvement in road traffic accidents and alter quality of life. This systematic review focused on the role of nonabsorbable disaccharides for the management of minimal hepatic encephalopathy.
Donna M, Zucker, Rhoda, Redulla
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Advances in the Evaluation and Management of Minimal Hepatic Encephalopathy
Current Gastroenterology Reports, 2010Minimal hepatic encephalopathy (MHE) is a neurocognitive disorder that affects up to 80% of cirrhotic patients. Similar to overt hepatic encephalopathy, ammonia and oxidative stress play key roles in the pathogenesis of MHE. However, MHE is characterized by subtle deficits and psychomotor abnormalities that can only be elicited by specialized ...
Jennifer Y, Montgomery +1 more
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Diagnosis of Minimal Hepatic Encephalopathy
2012Minimal hepatic encephalopathy (MHE) is a neurocognitive disorder that can affect up to 80% of cirrhotic patients. Patients seem clinically unimpaired, but have subtle deficits and psychomotor abnormalities that can only be elicited by specialized tests.
Jennifer Y. Montgomery +1 more
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Rifaximin and Minimal Hepatic Encephalopathy
American Journal of Gastroenterology, 2011Minimal hepatic encephalopathy (MHE) occurs in up to 70% of patients with cirrhosis and has a clear impact on health-related quality of life (HRQOL) in these patients. Antibiotics leading to reductions in circulating ammonia have been used in the past for the treatment of MHE.
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Diagnosis of minimal hepatic encephalopathy: still a challenge
Gut, 2013Hepatic encephalopathy (HE) describes the spectrum of potentially reversible neuropsychiatric abnormalities seen in patients with liver dysfunction after exclusion of unrelated neurological and/or metabolic abnormalities. There is a continuum of changes in brain function from ‘normal’ to overt HE.
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