Results 221 to 230 of about 2,014,208 (347)

Molecular marker aided breeding for blast resistant rice in Nepal

open access: bronze, 2014
Bal K Joshi   +3 more
openalex   +2 more sources

CD47 promotes mitogen‐activated protein kinase and epithelial‐to‐mesenchymal transition molecular programs to drive prometastatic phenotypes in non‐small cell lung cancer

open access: yesMolecular Oncology, EarlyView.
Beyond its role in immune evasion, this study identified that CD47 drives tumor‐intrinsic signaling in non‐small cell lung cancer (NSCLC). Transcriptomic profiling and functional studies revealed that CD47 regulates cell adhesion, migration, and metastasis through an ERK–EMT signaling axis.
Asa P.Y. Lau   +8 more
wiley   +1 more source

Development and application of a <i>Puccinia triticina</i> avirulence gene <i>AvrLr15</i>-specific molecular marker. [PDF]

open access: yesFront Plant Sci
Jin Y   +11 more
europepmc   +1 more source

Genomics and Molecular Markers for Major Cucurbitaceae Crops

open access: diamond, 2015
Girim Park, Nahui Kim, Young-Hoon Park
openalex   +1 more source

KDM7A and KDM1A inhibition suppresses tumour promoting pathways in prostate cancer

open access: yesMolecular Oncology, EarlyView.
Treatment resistance is a major challenge for patients with advanced prostate cancer. This study examined an alternative approach to target the major prostate cancer‐promoting pathway by targeting epigenetic factors, whose levels are higher in tumours.
Jennie N Jeyapalan   +16 more
wiley   +1 more source

Towards the identification of retrotransposon-based and ISSR molecular markers associated with populations resistant to ZYMV in melon

open access: hybrid, 2015
Babak Abdollahi Mandoulakani   +5 more
openalex   +1 more source

Cell‐cycle‐specific lesion evolution rather than inhibition of double‐strand‐break repair underpins cisplatin radiosensitization

open access: yesMolecular Oncology, EarlyView.
We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu   +10 more
wiley   +1 more source

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