Results 241 to 250 of about 5,528,146 (299)

The Neuroendocrine Response to Multiple Trauma

Journal of Urology, 1983
AbstractThe neuroendocrine response to multiple trauma is a coordinated, complex, changing response which has as its objective maintenance of life by preserving oxygen delivery and the mobilization and utilization of the synthetic and energetic substrates required by the body.The afferent system is composed of neural input from the various chemoceptors,
D S, Gann, M P, Lilly
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Complications of Multiple Trauma

Critical Care Nursing Clinics of North America, 1989
Patients who acquire sepsis, ARDS, ARF, or MSOF subsequent to multiple trauma have a high mortality rate. The pathophysiology of these complications is complex and is thought to involve ischemia, the generation of mediators, alterations in regional perfusion, and cellular oxygen use.
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Management of Multiple Trauma

Pediatric Clinics of North America, 1985
The first 20 minutes of medical treatment are crucial in determining the outcome for the multiple injured child. The author chronicles management procedures and their sequence, emphasizing the systemic approach and preparation as important tools.
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Multiple trauma

2014
AbstractCase 8.1 describes in detail the assessment and management of a multiply injured patient from their arrival in the emergency department through to their intraoperative period and postoperative care. Challenges encountered at every stage are discussed and key learning points explored.
Caroline Sampson, Michael Parr
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Trauma and multiple sclerosis

Journal of Neurology, 1987
An obligatory event in the pathogenesis of the multiple sclerosis plaque appears to be an increase in the permeability of the blood-brain barrier. Neuropathological observations of the brain of persons suffering from concussion after relatively minor head injury, as well as of animals subjected to experimental brain injury, have shown that alterations ...
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Thromboembolism Following Multiple Trauma

The Journal of Trauma: Injury, Infection, and Critical Care, 1992
The true incidence of thromboembolic complications following multiple trauma is unknown, and no method of prophylaxis has been shown to be both safe and effective in managing seriously injured patients. In this prospective study, 113 trauma patients were assigned on admission to receive either low-dose heparin (LDH), (5,000 U subcutaneously every 12 ...
M M, Knudson, J A, Collins, S B, Goodman, D W, McCrory   +3 more
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Acute trauma with multiple injuries

Current Opinion in Anaesthesiology, 2001
Trauma with multiple injuries is a leading cause of death. It presents a diversity of challenges and requires many healthcare workers to care for its victims. Advances continue in the organization of pre-hospital care, the techniques of trauma surgery and critical care, and understanding the pathophysiology of traumatic injuries.
R E, Johnstone, D F, Graf
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[Facial trauma and multiple trauma].

Soins; la revue de reference infirmiere, 2014
The human face contains the sense organs and is responsible for essential functions: swallowing, chewing, speech, breathing and communication. It is also and most importantly the seat of a person's identity. Multiple trauma adds a life-threatening dimension to the physical and psychological impact of a facial trauma.
Pierre, Corre, Ludovic, Arzul, Roman Hossein, Khonsari, Jacques, Mercier   +3 more
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The Septic Multiple-Trauma Patient

Critical Care Clinics, 1988
Sepsis in the multiple trauma patient is being seen with increased frequency now that more of these patients are surviving the initial period. Traumatic destruction of tissue barriers, the placement of various tubes and drains, and surgical repair with debridement all provide conduits for colonization and infection with pathogens.
M, Stillwell, E S, Caplan
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Hypercoagulability following Multiple Trauma

World Journal of Surgery, 1996
AbstractWe sought evidence of hypercoagulability in 59 seriously injured trauma patients. An extended coagulation profile (consisting of tissue plasminogen activator antigen concentration, plasminogen activator inhibitor, serum antithrombin III, protein C antigen, functional protein C, protein S antigen, D‐dimer, and prothrombin fragment 1.2) was ...
D T, Engelman, S G, Gabram, L, Allen, G E, Ens, L M, Jacobs   +4 more
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