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Patient rights in precision oncology: right treatment, right time, right dose, right order, right place. [PDF]
Fountzilas E+2 more
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The first report of pseudoaneurysm secondary to Cunninghamella bertholletiae infection in a Haematopoietic stem cell transplantation recipient: a case report and literature review. [PDF]
Ding Z+8 more
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Bosutinib-Induced Pleural Effusion-Class Effect and Cross-Intolerance to All Tyrosine Kinase Inhibitors. [PDF]
Vojjala N+7 more
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Acceptability and feasibility of acceptance and commitment therapy for improving outcomes in hematopoietic stem cell transplant. [PDF]
Merwin RM+12 more
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Statement of Retraction: MYCN contributes to the sensitization of acute myelogenous leukemia cells to cisplatin by targeting SRY-box transcription factor 4. [PDF]
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ASH Image Bank, 2001
Chronic myelogenous leukemia (CML) is a clonal hematopoietic stem cell disorder characterized by the (9:22) translocation and resultant production of the constitutively activated bcr-abl tyrosine kinase. Characterized clinically by marked myeloid proliferation, it invariably terminates in an acute leukemia.
Brian J. Druker, Michael J. Mauro
+11 more sources
Chronic myelogenous leukemia (CML) is a clonal hematopoietic stem cell disorder characterized by the (9:22) translocation and resultant production of the constitutively activated bcr-abl tyrosine kinase. Characterized clinically by marked myeloid proliferation, it invariably terminates in an acute leukemia.
Brian J. Druker, Michael J. Mauro
+11 more sources
Current Opinion in Hematology, 1995
Chronic myelogenous leukemia involves clonal expansion of hematopoietic progenitor cells associated with the characteristic translocation between chromosomes 9 and 22, resulting in the generation of an aberrant bcr-abl protein with enhanced tyrosine kinase activity.
Helen Enright, Philip B. McGlave
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Chronic myelogenous leukemia involves clonal expansion of hematopoietic progenitor cells associated with the characteristic translocation between chromosomes 9 and 22, resulting in the generation of an aberrant bcr-abl protein with enhanced tyrosine kinase activity.
Helen Enright, Philip B. McGlave
openaire +4 more sources