Results 331 to 340 of about 1,121,875 (382)
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Localized disorders in myocardial contraction. Asynergy and its role in congestive heart failure.
New England Journal of Medicine, 1967CONGESTIVE heart failure has usually been considered to be a global affection of the myocardium in which disturbance of contraction in one or both ventricles is generalized.
M. Herman +6 more
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Effect of vanadate on myocardial force of contraction
Basic Research in Cardiology, 1980Ammonium vanadate (NH4VO3; 50-1000 microM) increases the force of contraction of isolated electrically driven cat papillary muscles in a concentration-dependent manner. The positive inotropic effect (PIE) of NH4VO3 became significant at 50 microM and was maximal at 500 to 1000 microM.
Wilhelm Schmitz +3 more
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Acta Physiologica Scandinavica, 1968
Edman, K. A. P. and E. Nilsson. The mechanical parameters of myocardial contraction studied at a constant length of the contractile element. Acta physiol. scand. 1968. 72. 205–219.
K. Edman, E. Nilsson
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Edman, K. A. P. and E. Nilsson. The mechanical parameters of myocardial contraction studied at a constant length of the contractile element. Acta physiol. scand. 1968. 72. 205–219.
K. Edman, E. Nilsson
semanticscholar +1 more source
Reversible myocardial contraction abnormalities in patients with an acute noncardiac illness.
Chest, 1998STUDY OBJECTIVES Reversible myocardial contraction abnormalities are usually observed in patients with acute ischemic syndromes caused by coronary artery disease.
S. Sharkey +3 more
semanticscholar +1 more source
Myocardial protection: an expanding or contracting discipline?
Perfusion, 2003For the past thirty years cardiac surgeons have had a unique opportunity to study the consequences of myocardial ischemia and reperfusion. With the advent of an ability to intervene in acute myocardial syndromes, cardiologists have vigorously joined that effort as have fundamental scientists.
Andrew S. Wechsler, Stanley K. Brockman
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Identification of a PKCε-dependent regulation of myocardial contraction by epicatechin-3-gallate
, 2008In this study, the effects of tea catechins and tea theaflavins on myocardial contraction were examined in isolated rat hearts using a Langendorff-perfusion system.
Daxiang Li +7 more
semanticscholar +1 more source
Cardiovascular Research, 1993
OBJECTIVE Nitric oxide released by a calcium dependent constitutive NO synthase in endocardial endothelial cells exerts characteristic effects on myocardial contraction.
H. Evans, Malcolm J. Lewis, A. M. Shah
semanticscholar +1 more source
OBJECTIVE Nitric oxide released by a calcium dependent constitutive NO synthase in endocardial endothelial cells exerts characteristic effects on myocardial contraction.
H. Evans, Malcolm J. Lewis, A. M. Shah
semanticscholar +1 more source
American Review of Respiratory Disease, 1987
Myocardial contraction band necrosis (MCBN) occurs in catecholamine infusion, central nervous system stimulation, stress, and transient myocardial ischemia with reperfusion. In 4 of 13 children who died with asthma, MCBN was present, suggesting that this
F. Drislane +4 more
semanticscholar +1 more source
Myocardial contraction band necrosis (MCBN) occurs in catecholamine infusion, central nervous system stimulation, stress, and transient myocardial ischemia with reperfusion. In 4 of 13 children who died with asthma, MCBN was present, suggesting that this
F. Drislane +4 more
semanticscholar +1 more source
A Note on the Influence of Asynchronous Activation on Myocardial Contraction
Acta Physiologica Scandinavica, 1972AbstractPairs of rabbit papillary muscles were mounted in series to make it possible to study the effects of asynchronous activation on myocardial contraction. The two muscles were connected together via platinum loops hooked to a lever (displacement transducer), the other end of each muscle being fixed to a force transducer.
M. Jóhannsson, Erik Nilsson
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Physiology of Myocardial Contraction
2002The principal function of the heart is to propel oxygenated blood to the peripheral tissues to meet their metabolic demands. The systemic arterial and venous systems provide the conduits. The interaction of the left ventricle (LV) with the arterial and venous systems is therefore integral to the satisfactory performance of this vital function.
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