Abstract Background and aims The heart is a metabolic organ rich in mitochondria. The failing heart reprograms to utilize different energy substrates, which increase its oxygen consumption. These adaptive changes contribute to increased oxidative stress.
Qinghong Li+12 more
wiley +1 more source
IGFBP5 mediates the therapeutic effect of isoliquiritigenin in myocardial ischemia-reperfusion injury via AKT/GLUT4 regulated insulin resistance. [PDF]
Bai J+8 more
europepmc +1 more source
Effects of STAT4 on myocardial ischemia‑reperfusion injury and the underlying mechanisms. [PDF]
He M, Yu Y, Ning S, Han J, Guo Z.
europepmc +1 more source
Vaspin inhibits ferroptosis: A new hope for treating myocardial ischemia-reperfusion injury.
Lin X, Xin L, Meng X, Chen D.
europepmc +1 more source
AI-enabled diagnosis and localization of myocardial ischemia and coronary artery stenosis from magnetocardiographic recordings. [PDF]
Tao R+10 more
europepmc +1 more source
Preconditioning with acteoside ameliorates myocardial ischemia‑reperfusion injury by targeting HSP90AA1 and the PI3K/Akt signaling pathway. [PDF]
Li J+7 more
europepmc +1 more source
From smog to scarred hearts: unmasking the detrimental impact of air pollution on myocardial ischemia-reperfusion injury. [PDF]
Pota P+3 more
europepmc +1 more source
Semaglutide attenuates myocardial ischemia-reperfusion injury by inhibiting ferroptosis of cardiomyocytes via activation of PKC-S100A9 axis. [PDF]
Liu Y+7 more
europepmc +1 more source
ESC Heart Failure, Volume 12, Issue 2, Page 1514-1522, April 2025.
Takamasa Iwai+4 more
wiley +1 more source