Results 271 to 280 of about 21,869 (297)
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Hypertension, Natriuresis, and the Renal Prostaglandins
Annals of Internal Medicine, 1969Excerpt Prostaglandins are a group of cyclical fatty acids with diverse potent biological activities which were discovered independently in the early 1930's by Goldblatt (1) and von Euler (2).
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Natriuresis and vascular tone in toxemia of pregnancy
American Journal of Obstetrics and Gynecology, 1960E v E N though the signs and symptoms of pre-eclampsia and eclampsia are well known, the etiology and exact pathophysiology remain obscure. Through meticulous prenatal and puerperal care by controlling the early manifestations of the disease, much has been accomplished in preventing the severe degrees of pre-eclampsia and eclampsia.
Russell R. de Alvarez, Jose F. Afonso
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Exaggerated Natriuresis in ACTH Hypertension in Sheep
Nephron, 1978Adrenocorticotrophic hormone (ACTH) administration to sheep (100 IU/day) produces a sustained increase in arterial pressure within 24 h. The effect of ACTH on excretion of an intravenous saline load was tested in 8 adult cross-bred Merino ewes. A significant diuretic response to saline loading was found after 24 h but no increased natriuresis.
John G. McDougall+5 more
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Natriuresis in Rats Acutely Depleted of Chloride
Clinical Science, 19771. Acute chloride depletion, without sodium depletion, was produced in rats by a single exchange peritoneal dialysis against sodium bicarbonate solution. Blood volume was restored after dialysis by infusion of salt-free albumin, and exogenous deoxycorticosterone and antidiuretic hormone were given. 2. Clearance studies in the period (3 h)
Robert G. Luke+3 more
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Cerebral involvement in dehydration-induced natriuresis
Brain Research, 1983Water deprivation caused daily urinary Na output to more than double in normal sheep, but caused no increase in Na excretion in sheep (A3VL-sheep) in which the anterior third ventricle wall had been ablated. Dehydrated A3VL-sheep exhibited a much greater degree of hypernatremia than dehydrated normal sheep, although water losses were similar in both ...
R. G. Park+3 more
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The Role of Prostaglandins in Glucagon-Induced Natriuresis
Clinical Science, 19801. Three groups of anaesthetized dogs were studied to determine the role of renal prostaglandins in glucagon-induced natriuresis. 2. Urine flow, sodium and prostaglandin E excretion rates increased significantly in the experimental kidney with glucagon infusion (0.20 μg/min) into the renal artery.
M. A. Kirschenbaum, E. T. Zawada
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Resistance to Natriuresis in Patients with Peritonitis Carcinomatosa
Hormone and Metabolic Research, 1996The natriuretic effect of atrial natriuretic peptide (ANP) is blunted in certain clinical disorders such as congestive heart failure and liver cirrhosis, despite the elevated plasma ANP levels. These sodium-retaining states are characterized by increased activity of the renal sympathetic nerves.
T. Ogawa+3 more
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Role of prostaglandins in captopril-induced natriuresis.
Proceedings of the European Dialysis and Transplant Association - European Renal Association. European Dialysis and Transplant Association - European Renal Association. Congress, 1985To determine whether prostaglandins (PG) contribute to captopril-induced natriuresis, 20 hypertensive subjects were assigned to one of the following three groups: group a, captopril (C) administration; group b, C + indomethacin (I); group c, I alone. Captopril was given in a dose of 100, 200, 400 mg/day and indomethacin in a dose of 100mg/day for one ...
G. CONTE+7 more
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Mechanisms of angiotensin II natriuresis and antinatriuresis
American Journal of Physiology-Renal Physiology, 1985The aim of this study was to determine the role of changes in renal arterial pressure (RAP), renal hemodynamics, and tubular reabsorption in mediating the natriuretic and antinatriuretic actions of angiotensin II (ANG II). In seven anesthetized dogs, endogenous ANG II formation was blocked with captopril, and ANG II was infused intravenously at rates ...
J. E. Cornell+5 more
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Natriuresis after Preoptic Lesions in Rats
Canadian Journal of Physiology and Pharmacology, 1972Bilateral lesions in the preoptic area of the rat hypothalamus were followed by an increased renal excretion of sodium. This natriuretic response usually lasted several hours and in some animals the peak rate of excretion was as much as 100 times the control rate.The response was not prevented by large doses of desoxycorticosterone acetate, removal of
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