Results 121 to 130 of about 947 (163)
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Nesidioblastosis in a Simmental Calf

Journal of Comparative Pathology, 2012
A 2-day-old Simmental calf with arthrogryposis and astasia was subjected to necropsy examination. The calf was normoglycaemic and normoinsulinaemic. Microscopically, pancreatic tissue was hyperplastic with an irregular lobular arrangement of pancreatic islets.
Gacar, A.   +4 more
openaire   +3 more sources

Nesidioblastosis in Adults

Archives of Surgery, 1981
Nesidioblastosis is the leading cause of hyperinsulinemia in newborns and infants. To our knowledge, it has not been previously reported in adults unless associated with other diseases. Three males and three females, aged 11 to 57 years, are described. Pancreatic resections ranged from 50% to 100%.
J K, Harness   +7 more
openaire   +2 more sources

Beta cell nesidioblastosis

European Journal of Pediatrics, 1978
Two patients with severe hypoglycemia since birth are described. In both hyperinsulinism was demonstrated during spontaneous hypoglycemic attacks or could be provoked by various tolerance tests. In case I considerable obesity and psychomotor retardation was present at the age of one year whereas in case II weight gain was normal and development ...
K, Becker   +5 more
openaire   +2 more sources

Hypoglycemia of Infancy and Nesidioblastosis

New England Journal of Medicine, 1977
We treated a two-month-old infant with servere intractable hypoglycemia and nesidioblastosis with continuous glucose infusions (0.75 g per kilogram per hour) via a central venous catheter. Preprandial glucose levels on this regimen were 37+/-2 mg per deciliter (+/-S.E.M.).
H J, Hirsch   +5 more
openaire   +2 more sources

Familial nesidioblastosis in two sisters

Surgery Today, 1995
We herein present two female siblings with persistent hyperinsulinemic hypoglycemia in the neonatal period who were diagnosed as having familial nesidioblastosis. Despite both the administration of diazoxide and the intravenous infusion of glucose, one of the affected infants died of severe metabolic acidosis at about 1 month of age, before ...
M, Yagi   +4 more
openaire   +2 more sources

Nesidioblastosis (diagnosis, surgical treatment)

Khirurgiya. Zhurnal im. N.I. Pirogova, 2015
Nesidioblastosis (NB) is rare disease with organic hyperinsulinism syndrome and caused by diffuse hyperplasia and/or hypertrophy of pancreatic islands of Langerhans.The results of surgical treatment of 3 patients with NB are presented. In all patients the diagnosis was suspected at the preoperative stage and confirmed by histological examination later.
A G, Kriger   +8 more
openaire   +2 more sources

Nesidioblastosis with necrotising enterocolitis: an enigma

Pediatric Surgery International, 2000
Necrotising enterocolitis with nesidioblastosis is a rare, lethal coexistence during the newborn period. We report this unusual combination leading to surgery: a partial pancreatectomy and resection with primary anastomosis, and discuss the dilemma of the procedure of choice during therapy.
M S, Kulkarni, V A, Upadhyay
openaire   +2 more sources

Nesidioblastosis and the Zollinger-Ellison syndrome

The American Journal of Digestive Diseases, 1968
The most common cause of the Zollinger-Ellison syndrome is the over-production of a gastrin-like hormone by a non-beta islet cell neoplasm of the pancreas. Hyperplasia and neoformation of islets alone and in combination with neoplasia have been reported in association with the syndrome, but there has been little effort to implicate these findings in ...
R E, Brown, W J, Still
openaire   +2 more sources

Nesidioblastosis in Sickle Cell Disease

Fetal and Pediatric Pathology, 2001
Although the endocrine pancreas appears to play an important role in the pathophysiology of sickle cell disease, very little is known about the morphologic changes in this tissue. Our study was initiated to delineate the microscopic features of the endocrine pancreas in a large autopsy series of sickle cell hemoglobinopathies.
D E, Culberson   +6 more
openaire   +2 more sources

Nesidioblastosis of the Pancreas

American Journal of Clinical Pathology, 1990
D, Kumar, N E, Warner, T L, Fong
openaire   +2 more sources

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