Results 261 to 270 of about 841,514 (309)
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Neurotrophins and Neuronal Plasticity

Science, 1995
There is increasing evidence that neurotrophins (NTs) are involved in processes of neuronal plasticity besides their well-established actions in regulating the survival, differentiation, and maintenance of functions of specific populations of neurons.
Hans Thoenen, H Thoenen
exaly   +3 more sources

Plasticity of neuronal receptors

The FASEB Journal, 1989
This article describes ways in which receptors, key components of signal propagation through a synapse, can mediate changes in that propagation. Changes occur at four levels: in the signal‐transducing capability of a single receptor molecule, in the number of receptors per cell, in the subcellular placement of receptor molecules ...
W L, Klein   +3 more
openaire   +2 more sources

Adenosine and neuronal plasticity

Life Sciences, 1996
Adenosine is considered an important neuromodulator of the nervous system acting at pre-, post- and non-synaptic levels. In the present review we describe how adenosine modifies paired-pulse facilitation (PPF), posttetanic depression (PTD), long-term potentiation (LTP), long-term depression (LTD) and depotentiation at the hippocampus, and therefore how
A, de Mendonça, J A, Ribeiro
openaire   +2 more sources

Brain Aging and Neuronal Plasticitya

Annals of the New York Academy of Sciences, 1992
In the present paper three aspects of the aging processes and their physiopathological implications will be presented: neuronal death, the information handling capabilityof neurons, and the processes of interneuronal communication.
L. F. AGNATI   +7 more
openaire   +3 more sources

A Model of Neuronal Intrinsic Plasticity

IEEE Transactions on Autonomous Mental Development, 2011
Recent experimental results have accumulated evidence that the neurons can change their response characteristics to adapt to the variations of the synaptic inputs, which is the so-called neuronal intrinsic plasticity mechanism. In this paper, we present a new model on neuronal intrinsic plasticity. We first show that the probability distribution of the
Chunguang Li
exaly   +3 more sources

Hebb and homeostasis in neuronal plasticity

Current Opinion in Neurobiology, 2000
The positive-feedback nature of Hebbian plasticity can destabilize the properties of neuronal networks. Recent work has demonstrated that this destabilizing influence is counteracted by a number of homeostatic plasticity mechanisms that stabilize neuronal activity.
G G, Turrigiano, S B, Nelson
openaire   +2 more sources

Cellular and molecular neuronal plasticity

2015
The brain has the capability to adapt to function when tissue is compromised. This capability of adaptation paves the road to recovery and allows for rehabilitation after a traumatic brain injury (TBI). This chapter addresses neuroplasticity within the context of TBI.
Grace S, Griesbach, David A, Hovda
openaire   +2 more sources

Psychomotor stimulants and neuronal plasticity

Neuropharmacology, 2004
Considerable evidence suggests that neuroadaptations leading to addiction involve the same glutamate-dependent cellular mechanisms that enable learning and memory. Long-term potentiation (LTP) and long-term depression (LTD) have therefore become an important focus of addiction research. This article reviews: (1) basic mechanisms underlying LTP and LTD,
Marina E, Wolf   +3 more
openaire   +2 more sources

Neuronal plasticity and Parkinson disease

Molecular and Chemical Neuropathology, 1995
The data reviewed here demonstrate a capacity for neuronal plasticity in the aged human brain, even when affected by neurodegenerative disorders. However, given their location and environment, these morphological changes may have beneficial or deleterious consequences.
P, Anglade   +3 more
openaire   +2 more sources

Lipid Peroxides and Neuronal Plasticity

1999
Long-term potentiation (LTP) of synaptic transmission is a well defined form of neuronal plasticity. The induction of LTP at perforant path-dentate granule cell and Schaffer collateral-CA1 pyramidal cell synapses of the hippocampus is known to require influx of Ca2+ through postsynaptic N-methy1-D-aspartate (NMDA) receptors, while the expression of LTP
M, Nishiyama   +7 more
openaire   +2 more sources

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