Results 131 to 140 of about 282,476 (297)
Risk factors and prognosis of some actual congenital infections
Purpose: modeling the risk of congenital infections (СI) based on a comprehensive assessment of the results of laboratory monitoring of pregnant women and infants.
S. Kh. Kuyumch’yan +2 more
doaj
Course of the blood urea in newborn rats, pigs and kittens
R. A. McCance, Mary Otley
openalex +2 more sources
The Postnatal Synthesis of Fetal Hemoglobin. I. Some Studies in Newborn Goats Using Radioactive Fe59 and Glycine-2-C14 [PDF]
Henk Visser +2 more
openalex +1 more source
An integrated nanoparticle PEDOT@PMOF is constructed as a redox‐active nanozyme and electroactive nanofiller in a cell‐affinity hydrogel platform for acute MI treatment. It is demonstrated that MOF‐based nanozymes have good catalytic ability, and the incorporation of PEDOT increases the conductivity.
Shuyi He +6 more
wiley +1 more source
Blood chemistry studies of normal newborn infants
Albert W. Holman, Albeṙt Mathieu
openalex +1 more source
In contrast to the adult mammalian heart, which lacks regenerative capacity, the neonatal heart retains a transient regenerative capacity. In this study, high mobility group box 2 (HMGB2) is identified as a crucial protein driving heart regeneration based on comprehensive analyses using quantitative proteomics with tandem mass tag (TMT) labelling, RNA ...
Liu‐Hua Zhou +17 more
wiley +1 more source
Obstructive Jaundice in Haemolytic Disease of the Newborn Treated with Magnesium Sulphate [PDF]
N. V. O’Donohoe
openalex +1 more source
FMO2 Promotes Angiogenesis via Regulation of N‐Acetylornithine
This study identifies flavin‐containing monooxygenase 2 (FMO2) as a novel proangiogenic regulator in endothelial cells. Targeted FMO2 ablation impairs vessel sprouting, whereas its compensation potently enhances angiogenesis. Metabolomics and single‐cell sequencing reveal that FMO2 drives vascular growth via the N‐acetylornithine/ATF3/NOTCH1 axis ...
Jingyi Wang +15 more
wiley +1 more source
This study identifies osteocyte‐derived FGF9 as a mechanosensitive factor that impairs osteogenesis in the underdeveloped maxilla by inducing FGFR2 nuclear translocation in preosteoblasts. Mechanical stimulation reduces FGF9 secretion, relieving its inhibitory effect. These findings reveal a novel osteocyte–preosteoblast FGF9–FGFR2 axis in craniofacial
Yiwen Zhou +15 more
wiley +1 more source

