Results 161 to 170 of about 6,932,227 (258)
Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer
Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim +7 more
wiley +1 more source
The weight of words: Applying concreteness to elite media language. [PDF]
Snyder D.
europepmc +1 more source
Western News, October 31, 1996
Western News Volume 32, Number 30 was published October 31, 1996 by the Dept.
Western News
core
This study integrates publicly available transcriptomic datasets to identify molecular signatures associated with response to neoadjuvant chemoradiotherapy in locally advanced rectal cancer. By analyzing a combination of multiple cohorts with bioinformatics approaches, we reveal biological pathways and immune‐related features that may improve ...
Aleksandra Stanojevic +10 more
wiley +1 more source
News coverage of the proposed menthol cigarette ban in the United States (2022-2024): a content analysis. [PDF]
Wackowski OA +8 more
europepmc +1 more source
The VHL tumor suppressor at the crossroad of protein folding, aggregation, and cancer
Mutations, environmental stress, and chaperone dysfunction can destabilize pVHL, promoting its conversion from the native folded state into amyloid‐like assemblies. This transition may contribute to protein storage, cell dormancy, survival, and drug resistance.
Lara Abad +2 more
wiley +1 more source
How to Use-And How Not to Use-Public Forums in Research. [PDF]
Spichak S.
europepmc +1 more source
Dallas News original business office, undated.
Image of nine men standing in the Dallas News' original business office.Title from finding aid. Recto: [typewritten on label affixed to original photograph] Dallas News Original Business Office, Dallas ...
Unknown
core
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico +5 more
wiley +1 more source

