Results 311 to 320 of about 3,430,031 (362)
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Oxidized lipoproteins and nitric oxide
Current Opinion in Lipidology, 1996Nitric oxide has an important biological role as endothelium-derived relaxing factor, a key agent in the maintenance of normal vascular tone. It can also suppress lipoprotein oxidation, a potential anti-atherogenic property. However, in arteries subject to hypercholesterolemia or atherosclerosis, whereas nitric oxide synthesis is normal its biological ...
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Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor
Nature, 1987R. Palmer, A. Ferrige, S. Moncada
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1997
Apoptosis occurs during normal development of an organism but also plays an important role under pathophysiological conditions. Nitric oxide (NO) exhibits a double-edged role in apoptosis induction. Both pro- and antiapoptotic effects of NO have been demonstrated. The proapoptotic effects appear to be linked to pathophysiological conditions, where high
Judith Haendeler +2 more
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Apoptosis occurs during normal development of an organism but also plays an important role under pathophysiological conditions. Nitric oxide (NO) exhibits a double-edged role in apoptosis induction. Both pro- and antiapoptotic effects of NO have been demonstrated. The proapoptotic effects appear to be linked to pathophysiological conditions, where high
Judith Haendeler +2 more
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Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly.
American Journal of Physiology, 1996J. Beckman, W. Koppenol
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American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2002
bastron and kaloyanides([2][1]) had already investigated the effects of sodium nitroprusside in the isolated kidney 8 years before the postulate of an endothelium-derived relaxing factor was made by Furchgott and Zawadzki ([14][2]).
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bastron and kaloyanides([2][1]) had already investigated the effects of sodium nitroprusside in the isolated kidney 8 years before the postulate of an endothelium-derived relaxing factor was made by Furchgott and Zawadzki ([14][2]).
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Nitric oxide and glomerulonephritis
Seminars in Nephrology, 2004Glomerulonephritis is a common clinical condition that is caused by immune-mediated injury to the kidney and is characterized by dysfunction of the glomerular capillary filtration barrier. Nitric oxide (NO), a ubiquitous molecule with many biological functions throughout the body, has been evaluated as an inflammatory mediator in these circumstances ...
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Nitric oxide and macrophage function.
Annual Review of Immunology, 1997J. MacMicking, Q. Xie, C. Nathan
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Vascular endothelial cells synthesize nitric oxide from L-arginine
Nature, 1988R. Palmer, D. Ashton, S. Moncada
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