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Nutritional and metabolic rickets

The Indian Journal of Pediatrics, 1997
Nutritional rickets is caused by vitamin D deficiency due to lack of exposure to sunlight. Neonatal rickets occurs only in infants born to mothers with very severe osteomalacia. Calcium deficiency alone does not cause mineralisation defects. It only causes osteoporosis and secondary hyperparathyroidism with raised plasma, 1,25 (OH)2D and osteocalcin ...
M, Teotia, S P, Teotia
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Nutritional Rickets in Suburbia

Journal of the American College of Nutrition, 1998
Vitamin D deficiency continues to be a problem in pediatrics. This report presents four children, one Caucasian male and three African-American females aged 4 to 24 months who were treated for vitamin D deficiency rickets.One female was diagnosed in the Emergency Department during evaluation of a viral syndrome, another presented with hypocalcemic ...
M T, Pugliese   +3 more
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Nutritional Rickets

Pediatric Emergency Care, 2006
In the emergency department, opportunities exist for the emergency physician to make a diagnosis beyond the chief complaint. For example, an astute reader of pediatric radiographs may detect signs of rickets on plain films that are obtained for other reasons.
Laurie, Hickey   +2 more
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Nutritional rickets

Trends in Endocrinology & Metabolism, 1991
Nutritional rickets was once felt to be a disease of the past. In recent years, dietary patterns and the prevalence of unsupplemented breast feeding has led to a mild resurgence in rickets. The cases presented show a wide spectrum of nutritional disease, illustrating the common risk factors, the usefulness of vitamin-D metabolite assays in making a ...
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Pseudotumor cerebri and nutritional rickets

European Journal of Pediatrics, 1985
A bulging fontanelle due to benign increased intracranial pressure is not generally recognized as a manifestation of nutritional rickets but should be considered in the appropriate clinical setting. Two children who we saw presented with bulging anterior fontanelles were found to have pseudotumor cerebri in association with nutritional rickets.
A R, DeJong, C A, Callahan, J, Weiss
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Nutritional rickets in Calabar, Nigeria

Annals of Tropical Paediatrics, 1995
The features of 20 cases of nutritional rickets who presented at the University of Calabar Teaching Hospital, Calabar in south-eastern Nigeria over a 9-year period (January 1985-December 1993) are presented. The aim is to compare them with those in the only other reports from Nigeria between 18 and 42 years ago from Ibadan in the west of the country ...
E E, Ekanem, D E, Bassey, M, Eyong
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Nutritional Rickets Presenting with Myelofibrosis

The Indian Journal of Pediatrics, 2010
We describe the clinical course of a 10-month-old breastfed infant with rickets and associated myelofibrosis presenting with anemia and hepatosplenomegaly. Over the follow up, on therapeutic supplementation of vitamin D, child showed reduction in liver and spleen size along with improvement in rickets, anemia, growth and developmental parameters.
Bhanu Kiran, Bhakhri, Pradeep K, Debata
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Reemerging Nutritional Rickets

Archives of Pediatrics & Adolescent Medicine, 2005
Recent case reports highlight the resurgence of rickets in certain groups of breastfed infants. Infants residing in the North, irrespective of skin color, and dark-skinned African American infants residing anywhere in the United States are most vulnerable to nutritional rickets if they are exclusively breastfed past age 6 months without vitamin D ...
Rajakumar, K, Thomas, S
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Nutritional Rickets in San Diego

Archives of Pediatrics & Adolescent Medicine, 1987
Despite the ability of infants to synthesize vitamin D through exposure to sunlight, nutritional rickets occasionally develops in infants even in areas with perennially sunny climates. In San Diego, a 1-year-old breast-fed infant presented with classic signs of nutritional rickets.
I, Hayward, M T, Stein, M I, Gibson
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Nutritional rickets around the world

The Journal of Steroid Biochemistry and Molecular Biology, 2013
Nutritional rickets is a major public health problem in many countries of the world. The disease is characterized by deformities of the long bones, enlargement of the wrists and costochondral junctions, hypotonia and, in infants, craniotabes and delayed fontanelle closure.
openaire   +2 more sources

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