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Progressive external ophthalmoplegia
Clinical Ophthalmic Genetics and Genomics, 2022Ungsoo S. Kim +2 more
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Amitriptyline‐induced ophthalmoplegia
Neurology, 1981Total external ophthalmoplegia, unresponsive to caloric stimulation, was observed in a gravid woman who had ingested approximately 1.0 to 1.5 gm of amitriptyline. The intravenous administration of 4.0 mg physostigmine salicylate (PS) produced a revival of consciousness and reflex activity but had no appreciable effect on ocular motility.
R H, Spector, R, Schnapper
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Progressive external ophthalmoplegia
2023Progressive external ophthalmoplegia (PEO), characterized by ptosis and impaired eye movements, is a clinical syndrome with an expanding number of etiologically distinct subtypes. Advances in molecular genetics have revealed numerous pathogenic causes of PEO, originally heralded in 1988 by the detection of single large-scale deletions of mitochondrial ...
Michio, Hirano, Robert D S, Pitceathly
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Phenytoin‐induced ophthalmoplegia
Neurology, 1976Total external ophthalmoplegia was observed in five patients consequent to the oral or intravenous administration of phenytoin. Coincident with the ophthalmoplegia, the state of consciousness varied from drowsiness to coma and the blood levels of phenytoin ranged from 36 to 55 mug per milliliter.
R H, Spector +2 more
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Reversible ophthalmoplegia in CPEO
Australian and New Zealand Journal of Ophthalmology, 1995Abstract Purpose: To present a case of improvement of ocular motility in a patient with chronic progressive external ophthalmoplegia (CPEO) with Coenzyme Q10. Methods: Coenzyme Q10 300 mg daily was given for three years with a three‐day trial period of 200 mg daily after one year. Ocular ductions were measured by synoptophore.
I S, Dunlop, P, Dunlop
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Archives of Neurology, 1968
CONSIDERABLE interest has been focused on the entity of progressive external ophthalmoplegia (PEO) since the report of Kiloh and Nevin in 1951. 1 These authors were primarily concerned with the pathogenesis of the ocular muscle weakness, concluding that it was virtually always the result of a myopathic process.
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CONSIDERABLE interest has been focused on the entity of progressive external ophthalmoplegia (PEO) since the report of Kiloh and Nevin in 1951. 1 These authors were primarily concerned with the pathogenesis of the ocular muscle weakness, concluding that it was virtually always the result of a myopathic process.
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Postgraduate Medicine, 1991
Cranial neuropathy affecting the eyes is a possible symptom of type II diabetes. In this article, Dr Dickerman describes a case in which diabetes was diagnosed in a 63-year-old woman who presented with unilateral headache and eye pain.
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Cranial neuropathy affecting the eyes is a possible symptom of type II diabetes. In this article, Dr Dickerman describes a case in which diabetes was diagnosed in a 63-year-old woman who presented with unilateral headache and eye pain.
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Carbamazepine-Induced Ophthalmoplegia
Archives of Neurology, 1982Carbamazepine is a derivative of iminostilbene related to the tricyclic antidepressants. It was introduced as an anticonvulsant and was later found useful in the treatment of trigeminal, glossopharyngeal, tabetic, and postherpetic neuralgic pain. Side effects have included diplopia and nystagmus. 1 I report a case of external ophthalmoplegia associated
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