Results 231 to 240 of about 15,797 (250)
Some of the next articles are maybe not open access.
The prevalence of pancreatitis in organophosphate poisonings
Human & Experimental Toxicology, 2002Objective: The aim of this study was to evaluate the prevalence of pancreatitis in cases of organophosphate (OP) poisonings admitted to Yüzüncü Yõl University Teaching Hospital over an 18-month period. Materials and Methods: A total of 47 patients of acute poisoning with OP insecticides attended the Emergency Department of the Yüzüncü Yõl Medical ...
Noyan, T +5 more
openaire +4 more sources
Organophosphate and Carbamate Poisoning
Archives of Internal Medicine, 1994Organophosphate insecticides may cause serious poisoning either accidentally or by deliberate ingestion. Toxic symptoms are produced by acetylcholine accumulation at cholinergic receptors. Diagnosis is based on history of exposure or ingestion, symptoms and signs of cholinergic overactivity and a decrease in serum pseudocholinesterase levels. Following
Alwyn P. Foden +5 more
openaire +2 more sources
Prolonged toxicity of organophosphate poisoning
Critical Care Medicine, 1982A case of poisoning with a new organophosphate (fenthion) is reported in which the initial cholinergic crisis was delayed 5 days and recurred 24 days after ingestion. Psychosis was a persistent and sometimes singular manifestation. Because of the high lipid solubility of this pesticide, toxin analysis of repeated fat biopsies was an essential component
Douglas G. Merrill, Frederick G. Mihm
openaire +3 more sources
Diagnostic aspects of organophosphate poisoning
Toxicology, 2005Organophosphate (OP)-type chemical warfare agents (nerve agents) present a constant threat to the population. Sensitive and specific methods for the detection and verification of exposure to nerve agents are required for diagnosis, therapeutic monitoring, health surveillance and forensic purposes.
Marianne Koller +3 more
openaire +2 more sources
Hormonal consequences of organophosphate poisoning
Fundamental and Applied Toxicology, 1985The hormonal changes were estimated after poisoning with an extremely toxic organophosphate, soman (pinacolyl methylphosphonofluoridate). Soman produced a significant (p less than or equal to 0.05) increase in serum corticosterone, thyroxine, and triidothyronine concentrations at 3, 6, and 9 hr after poisoning.
openaire +3 more sources
Treatment of acute organophosphate poisoning
Medical Journal of Australia, 1991The biochemistry and pharmacology of poisoning by organophosphorus esters and carbamates are outlined and methods of diagnosis and treatment are discussed. Treatment consists of reduction of exposure by decontamination, induction of vomiting or gastric lavage; pharmacological treatment with atropine and oximes (pralidoxime and obidoxime); and ...
openaire +4 more sources
Role of Aliesterase in Organophosphate Poisoning
Toxicological Sciences, 1984Various doses of CBDP (2-(2- methylphenoxy )-4H-1,3,2- benzodioxaphosphorin -2-oxide), a metabolite of tri-o-cresyl phosphate, increased dramatically the acute toxicity of soman ( pinacolyl methylphosphonofluoridate ) in mice. CBDP (5 mg/kg; iv) reduced the soman LD50 value from 136 micrograms/kg in control to 6.95 micrograms/kg.
openaire +3 more sources
Chapter 13 Organophosphate and carbamate poisoning
2008Publisher Summary Highly toxic acetylcholinesterase (AChE)-inhibiting organophosphates (OPs) and carbamates (CMs) are used as pesticides throughout the world. Most OPs are currently used as pesticides, paraciticides in veterinary medicine, warfare agents, or flame retardants. However, carbamates are used as pesticides in human and veterinary medicine
openaire +3 more sources