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Oromandibular dystonia

Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology, 2007
Oromandibular dystonia (OMD) is a rare neuromuscular disorder characterized by involuntary repetitive muscular contraction affecting different parts of the oromandibular region. Its various physical manifestations can be extremely debilitating and socially disabling to affected patients. To date, there is no commonly accepted set of diagnostic criteria
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Clozapine Treatment in Oromandibular Dystonia

Clinical Neuropharmacology, 2004
Oromandibular dystonia (OMD) is a form of focal dystonias, which can be associated with substantial disability and is frequently refractory to all antidystonic therapies. Clozapine is a dibenzodiazepin derivative atypical neuroleptic that has been reported to be effective in the treatment of primary or symptomatic dystonia.
Hasmet A, Hanagasi   +3 more
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Somatosensory input and oromandibular dystonia

Clinical Neurology and Neurosurgery, 2013
Oromandibular dystonia (OMD) involving the lateral pterygoid uscles (LPs) may cause incapacitating protrusive and lateral jaw ovements, but can be alleviated with botulinum toxin (BTX) [1]. ystonia is thought to be derived from dysfunction of the basal anglia, and the excess movement to be due to loss of inhibitory otor control.
Møller, E   +6 more
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Oromandibular dystonia: a diagnosis not to miss

British Journal of Oral and Maxillofacial Surgery, 2020
Oromandibular dystonia (OMD) is characterised by sustained or repetitive involuntary movements of the jaw, face, and tongue. People with the condition may present to their dentist, general practitioner, or a secondary care specialist with non-specific symptoms including jaw or facial pain, bruxism, subluxations or dislocations of the jaw; fractured ...
D. Britton, J.E. Alty, C.J. Mannion
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PATHOPHYSIOLOGY OF BLEPHAROSPASM AND OROMANDIBULAR DYSTONIA

Brain, 1985
The pathophysiology of reflexes mediated by the fifth and seventh cranial nerves has been studied in 16 patients with blepharospasm and oromandibular dystonia compared with normal age-matched subjects. The EMG activity of the dystonic spasms in the periocular and jaw muscles was similar to that described in other muscles in patients with generalized ...
BERARDELLI, Alfredo   +3 more
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[Therapeutic strategies for oromandibular dystonia].

Fortschritte der Neurologie-Psychiatrie, 2021
Oromandibular dystonia is characterized by tonic or clonic involuntary spasms of the masticatory, lingual and / or muscles in the stomatognathic system. It is often misdiagnosed as craniomandibular dysfunction or psychiatric disease. According to clinical features, the oromandibular dystonia is classified into 6 subtypes (jaw closing-, jaw opening ...
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Treatment of Oromandibular Dystonia

2019
Oromandibular dystonia (OMD) is a rare form of dystonia that affects the masticatory, lower facial, and lingual muscles. OMD is often idiopathic and can present as a focal dystonia or as part of a segmental or generalized pattern of dystonia. The movement phenotypes of OMD include jaw opening, jaw closing, lateral jaw deviation, lingual protrusion, and
Stewart A. Factor, Laura M. Scorr
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Smothering dystonia in a patient with oromandibular dystonia

Movement Disorders, 2004
AbstractA case report is presented of a patient with pathologically confirmed striatonigral degeneration who experienced episodic syncope as a result of oromandibular dystonia obstructing inhalation through her mouth and nose.
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Oromandibular Dystonia in Cerebrotendinous Xanthomatosis

The Laryngoscope, 2009
She was treated with speech therapy and serial titrated electromyography-guided BTX injections (Figure 5). A maximum of 15 units of BTX were administered to each of the external pterygoid muscles and 5 units of toxin were administered to each anterior belly of the digastric muscles.
Brian E. Benson   +2 more
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Pharmacology of blepharospasm‐oromandibular dystonia syndrome

Neurology, 1980
Blepharospasm and oromandibular dystonia are clinically similar to other hyperkinetic movement disorders. Dopaminergic antagonist (neuroleptic) and purported cholinergic agonist (deanol) treatment improved symptoms, whereas dopaminergic agonist (carbidopa/levodopa) and cholinergic antagonist (benztropine) drugs worsened symptoms in two patients.
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