Results 271 to 280 of about 179,681 (354)
Characteristics of out-of-hospital cardiac arrest due to cerebrovascular disorders: a nationwide, retrospective, observational study. [PDF]
Ueda Y +8 more
europepmc +1 more source
Disproportionality analyses suggested a cardiovascular risk signal for romosozumab, while statistically significant associations were not found in the real‐world database studies. Therefore, a larger comparative study was necessary to examine this signal.
Hotaka Maruyama +5 more
wiley +1 more source
Assessing adult attachment after out-of-hospital cardiac arrest: an exploratory analysis and construct validation of the ECR-RS. [PDF]
Hansen N +8 more
europepmc +1 more source
Few studies have quantified the effects of non‐pharmacological interventions (e.g., rescue breathing) in the setting of community opioid overdose. We extended a previously validated model for opioid antagonists by incorporating the mechanism of rescue breathing, and quantified combinatorial effects between rescue breathing and various formulations of ...
Bradlee Thrasher +12 more
wiley +1 more source
Effect of tele-support on attention levels of emergency medical technicians during simulated out-of-hospital cardiac arrest. [PDF]
Scharner V +11 more
europepmc +1 more source
Major Adverse Cardiac Events with Ondansetron: A Systematic Review
Evidence base: 170 randomized trials, 23,421 adults, predominantly single‐dose IV ondansetron, mean follow‐up 20 days. Observed outcomes: Major adverse cardiac events were rare, no ventricular arrhythmias or torsades de pointes observed. Conclusion: Ondansetron was not associated with increased mortality or serious cardiac arrhythmias in adult ...
Michael Cristian Garcia +11 more
wiley +1 more source
Extracellular mitochondrial DNA activates complement and is associated with complement activation in patients with out-of-hospital cardiac arrest. [PDF]
de Boer E +11 more
europepmc +1 more source
Abstract Background Sporadic venous malformation (VM) is associated with the hyperactivating p.L914F mutation in TIE2, a receptor tyrosine kinase essential for vascular development. This mutation is not found in hereditary VM, suggesting incompatibility with life when expressed during early vascular development.
Lindsay J. Bischoff +6 more
wiley +1 more source

