Results 121 to 130 of about 257,150 (173)
A Case of a Hemodialysis Patient With Myelodysplastic Neoplasm-Like Anemia Due to Hypozincemia Treatment. [PDF]
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[Pelger-Huet's nuclear anomaly; case report].
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Congenital Pelger-Huet Anomaly in Triplets
American Journal of Hematology, 1988AbstractCongenital Pelger‐Huet anomaly is an autosomal dominant disorder characterized by incomplete nuclear segmentation of granulocytes. The morphology of these mature but hyposegmented granulocytes may be confused with that of immature granulocytes (bands), thereby suggesting bacterial infection.
Russell E Ware +2 more
exaly +5 more sources
Acquired Pelger-Huet Anomaly in Erythroleukemia
Journal of Surgical Oncology, 1970AbstractThe Pelger‐Huet anomaly was found in association with erythroleukemia in a 62‐year‐old female who was known to have had normal granulocyte morphology 9 months previously. This is the third reported case of the coexistence of Pelger‐Huet anomaly with erythroleukemia and the first case in which normal morphology was known to have been present ...
Bruce A Phillips
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Pelger—Huet Anomaly in an Infant With Multiple Congenital Anomalies
American Journal of Hematology, 1991AbstractWe report a new case of Pelger‐Huet anomaly (PHA) evident from the first day of life in an infant with multiple congenital anomalies suggestive of Fryn syndrome. The infant's parents are not affected by PHA, raising the possibility that the PHA resulted from a spontaneous mutation.
Julie D Fishbein, John M Falletta
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Pelger-Huet Anomaly of Granulocytes in a Patient With Tuberculosis
Chest, 1984The association of abnormal granulocyte nuclear formation and tuberculosis was first reported by Pelger in 1928. Huet, however, concluded this defect was hereditary, and the association of Pelger-Huet anomaly (PHA) of granulocytes and tuberculosis was obscured for many years.
R Phillip Dellinger, R P Dellinger
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Association of Acquired Pelger-Huet Anomaly With Taxoid Therapy
British Journal of Haematology, 1996We describe the occurrence of acquired Pelger‐Huet anomaly (APHA) in 23 patients treated with paclitaxel (13) or docetaxel (10). A consistent peak of Pelger‐Huet cells (PHC) within a range of 3–9d after treatment with taxoids was noted. The APHA generally disappeared by day 21 after treatment.
Guy Toner, Michael Michael, M J Millward
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Transfer of Pelger-Huet Anomaly by Bone Marrow Transplantation
American Journal of Hematology, 1982AbstractA marrow transplant was performed on a patient with refractory acute myelomonocytic leukemia using marrow from a sibling donor with congenital Pelger‐Huet anomaly (P‐H). The patient, free of P‐H prior to transplant, subsequently acquired the anomaly, thus providing a useful means of demonstrating engraftment of donor cells.
E D Thomas
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Incidence of Infections in Patients With Pelger-Huet Leucocyte Anomaly.
British Journal of Haematology, 1980The Pelger‐Huet (PH) leucocyte anomaly is manifested by the presence in the peripheral blood of granulocytes with a non‐lobulated nucleus, but it is generally assumed that this has no clinical effects. However, by careful observation of a group of 85 subjects (from 32 families) with the PH anomaly the incidence of infections, particularly of local ...
J Aznar
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Pelger-Huet Anomaly: A Rare Case Report
Pelger-Huet anomaly (PHA) is a rare benign genetic disorder affecting terminal differentiation of granulocytes. Granulocytes have hyposegmented nuclei with normal cytoplasmic granularity and perform immune function.
Bhagyalakshmi Atla
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