Results 181 to 190 of about 357,951 (342)
Assessment of the backscattered skin dose from the beam stopper on Halcyon and Ethos radiotherapy systems. [PDF]
Lankinen L +4 more
europepmc +1 more source
Some phantom designs for radiation dosimetry and CT applications [PDF]
Bruce Thomadsen +4 more
openalex +1 more source
β‐TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1
Low levels of β‐TrCP (Panel A) allow the accumulation of BRCA1 and CtIP, which facilitate the repair of cisplatin‐induced DNA damage via homologous recombination (HR) and promote tumor cell survival. In contrast, high β‐TrCP expression (Panel B) leads to BRCA1 and CtIP degradation, impairing HR repair, resulting in persistent DNA damage and apoptosis ...
Rocío Jiménez‐Guerrero +8 more
wiley +1 more source
MR elastography datasets including phantom, liver, and brain. [PDF]
Feng Y +5 more
europepmc +1 more source
THE TRIAL OF A JAW PHANTOM PRODUCED AT OUR INSTITUTES
Huminori NAKAO +11 more
openalex +2 more sources
Stemness properties, including quiescence, self‐renewal, and chemoresistance, are closely associated with leukemia relapse. Here, we demonstrate that DNA damage‐inducible transcript 4 (DDIT4) is induced in the hypoxic bone marrow niche and is essential for maintaining the stemness of AML1‐ETO9a leukemia cells.
Yishuang Li +12 more
wiley +1 more source
Segmental airway adenocarcinoma-simulating phantom for endoscopic near-infrared optical coherence tomography. [PDF]
Brace E +5 more
europepmc +1 more source
Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs
In HGSOC with normal KRAS expression, high PTP4A3 expression regulates autophagy activation. Conversely, in HGSOC with high KRAS expression, KRAS dictates autophagy control, and PTP4A3 is not required. When high PTP4A3 expression is inhibited, HGSOC cells are preferentially sensitised towards DNA‐damaging agents.
Ana López‐Garza +3 more
wiley +1 more source

