Results 311 to 320 of about 382,869 (342)
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PATHOPHYSIOLOGY OF PLAQUE RUPTURE AND THE CONCEPT OF PLAQUE STABILIZATION
Cardiology Clinics, 1996Atherosclerotic coronary artery disease is the major cause of death, in men and women, in the United States and in much of the Western world. Atherosclerosis is responsible for coronary heart disease, limb ischemia, and most strokes. Although luminal narrowing by an atherosclerotic plaque and exaggerated or anomalous vasoconstriction contribute to some
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Plaque regrowth studies: discriminatory power of plaque index compared to plaque area
Journal of Clinical Periodontology, 1999Abstract. A large number of plaque‐scoring methods exist for the evaluation of plaque inhibition by oral hygiene agents, products or devices. The ability of such methods to discriminate between high and low plaque scores has not been compared. In this retrospective analysis, the F‐ratios for differences between treatments for plaque index and area ...
Martin Addy +2 more
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Cerebrovascular Diseases, 2004
Ischaemic strokes and transient ischaemic attacks are commonly caused by cerebral embolism originating from formation of a platelet-rich thrombus superimposed on an atherosclerotic plaque or by atherothrombotic plaque rupture in a carotid or intracranial artery.
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Ischaemic strokes and transient ischaemic attacks are commonly caused by cerebral embolism originating from formation of a platelet-rich thrombus superimposed on an atherosclerotic plaque or by atherothrombotic plaque rupture in a carotid or intracranial artery.
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HISTOPATHOLOGY OF PLAQUE RUPTURE
Cardiology Clinics, 1999Plaque disruption occurs during the development of atherosclerotic lesions. During certain circumstances it may result in thrombosis and subsequent development of acute coronary syndromes. Several characteristics of the plaque appear to be associated with plaque disruption, including a large lipid rich core, superficial plaque inflammation, and a thin ...
Ravn, Hanne Berg, Falk, Erling
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Science Signaling
Early exposure to cholesterol primes later atherosclerosis through phenotypic changes to resident-like arterial macrophages.
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Early exposure to cholesterol primes later atherosclerosis through phenotypic changes to resident-like arterial macrophages.
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The International Journal of Cardiovascular Imaging, 2008
Stephen J. Green +2 more
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Stephen J. Green +2 more
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A Non‐Invasive Nanoprobe for In Vivo Photoacoustic Imaging of Vulnerable Atherosclerotic Plaque
Advanced Materials, 2020Lemin Zheng, Jian Kong, Lingyun Zu
exaly