Results 251 to 260 of about 4,625,362 (300)

Pregnancy‐induced hypertension in twin pregnancy

BJOG: An International Journal of Obstetrics & Gynaecology, 1984
summaryA prospective study of 54 primigravid twin pregnancies in Durban showed that pregnancy‐induced hypertension occurred in 37%. This was more than twice the expected rate for singleton pregnancies in this Black African population. The babies born in the group of patients with pregnancy‐induced hypertension were significantly lighter and had a ...
P F, McMullan, R J, Norman, M, Marivate
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Pregnancy-induced hypertensive disorders

Current Opinion in Obstetrics and Gynecology, 1994
The concept that endothelial cell activation and injury constitutes the key pathogenetic event is receiving increasing support, but the causative factor or factors are, as yet, unclear. The search for a reliable and clinically useful test to identify pregnant women at risk continues.
H C, Wallenburg, W, Visser
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Enalapril and Pregnancy-Induced Hypertension

Annals of Internal Medicine, 1988
Excerpt To the editor: Shubiger and colleagues (1) have confirmed that administering angiotensin-converting enzyme inhibitors to pregnant women can provoke renal failure in the newborn.
M D, Lindheimer, W M, Barron
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Anesthesia for Pregnancy-Induced Hypertension

Clinics in Perinatology, 1982
Women with severe pregnancy-induced hypertension are often critically ill; their fetuses are usually compromised. An ideal anesthetic method does not exist for the parturient with severe hypertension, hypovolemia, and organ failure. Optimal anesthetic results depend upon thorough preanesthetic evaluation and best medical control of pathophysiology ...
A S, Wheeler, B A, Harris
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The Endocrinology of Pregnancy-Induced Hypertension

Clinics in Perinatology, 1983
Discussed in this article are the effects of pregnancy-induced hypertension on the renin-angiotensin-aldosterone system; the possible role of prostanoids, deoxycorticosterone, and deoxycorticosterone sulfate in blood pressure control; and the effect of pregnancy-induced hypertension upon the endocrinology of the maternal-fetal-placental unit.
B R, Carr, N F, Gant
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Labetalol pharmacokinetics in pregnancy-induced hypertension

American Journal of Obstetrics and Gynecology, 1990
Pharmacokinetic parameters of oral labetalol were studied in eight women with pregnancy-induced hypertension in the third trimester of pregnancy. Labetalol exhibited rapid absorption; peak serum concentrations of 881 +/- 219 ng/ml occurred at 20 minutes after labetalol ingestion.
R C, Rogers, B M, Sibai, W D, Whybrew
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Neutrophil activation in pregnancy‐induced hypertension

BJOG: An International Journal of Obstetrics & Gynaecology, 1989
Summary. Human neutrophil elastase may be a major mediator of vascular damage and could contribute to the vascular damage seen in women with pregnancy‐induced hypertension (PTH). Elevated plasma levels of this substance will reflect neutrophil activation in vivo.
I A, Greer, N G, Haddad, J, Dawes, F D, Johnstone, A A, Calder   +4 more
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PLASMA PROLACTIN IN PREGNANCY INDUCED HYPERTENSION

BJOG: An International Journal of Obstetrics & Gynaecology, 1978
SummaryIn a prospective study, plasma prolactin concentration at 37 to 41 weeks gestation in 45 primigravidae showed a significant correlation with maximal rise in diastolic blood pressure between levels at 7 to 16 weeks and those measured between 28 weeks and delivery.
D M, Jenkins, L A, Perry
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On the Alert for Pregnancy-Induced Hypertension

Home Healthcare Nurse: The Journal for the Home Care and Hospice Professional, 2001
Increasing the skills of maternal-child home care nurses can identify early incidences of pregnancy-induced hypertension (PIH) for medical intervention, resulting in the prevention of hospitalization and decrease of potential complications. This article describes one agency's approach to teaching staff these important skills.
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Pregnancy-induced hypertension

1986
The best approach to the management of pregnancy-induced hypertension (PIH) would be its prevention (Table 17.1). Unfortunately, this goal will continue to elude us until we understand the primary pathogenesis of this condition. Some hypotheses are beginning to emerge.
F. Broughton Pipkin, E. M. Symonds
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