Results 261 to 270 of about 1,408,623 (295)
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Effect of acebutolol and propranolol on premature ventricular complexes
Clinical Pharmacology and Therapeutics, 1980A double-blind, randomized study comparing the efficacy of intravenous acebutolol with propranolol on frequent premature ventricular complexes (PVCs) in 24 patients is reported. Frequent PVCs were abolished or reduced by 75% or more in 10 of 12 patients (83%) given acebutolol and in 10 of 12 patients (83%) given propranolol.
W S, Aronow +4 more
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Ablation of frequent premature ventricular complex in an athlete
Scandinavian Journal of Medicine & Science in Sports, 2014Premature ventricular complex are common findings in the exam of many athletes. There is no extensive scientific evidence in the management of this situation particularly when associated with borderline contractile function of the left ventricle. In this case report, we present a 35‐year‐old asymptomatic healthy athlete with high incidence (over 10 000
G, Grazioli +5 more
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Premature Ventricular Beats with Narrow QRS Complexes
Acta Medica Scandinavica, 1976ABSTRACT A case is presented with premature ventricular beats with narrow QRS complexes. The arrhythmia is discussed on the basis of an ECG recording with 12 simultaneous ECG leads.
G, Forssell +3 more
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Ventricular Premature Complex Induced or Ventricular Premature Complex Worsened Cardiomyopathy
The American Journal of Cardiology, 2017Ozcan Ozeke +6 more
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Management of premature ventricular complexes.
Missouri medicine, 2010Premature ventricular contractions (PVCs) are frequently encountered, and management is determined by symptoms, precipitating factors, and the presence of underlying cardiac disease. No treatment is indicated in patients with asymptomatic PVCs in absence of cardiac disease.
Albert K, Chan, Mary L, Dohrmann
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MY APPROACH to the patient with premature ventricular complexes
Trends in Cardiovascular Medicine, 2016Frequent premature ventricular complexes (PVCs) can lead to a reversible cardiomyopathy. Frequent PVCs can also be a marker for the presence of structural heart disease. Both issues need to be addressed when a patient with frequent PVCs is evaluated. The PVC burden needs to be quantified and a predominant PVC morphology, if present, must be identified.
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