Results 251 to 260 of about 47,925,041 (381)

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

open access: yesMolecular Oncology, EarlyView.
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian   +10 more
wiley   +1 more source

Replicating the unconscious working memory effect: a multisite Registered Report. [PDF]

open access: yesNeurosci Conscious
Franco-Martínez A   +48 more
europepmc   +1 more source

The CHEK2 1100delC allelic variant is not present in familial and sporadic breast cancer cases from Moroccan population [PDF]

open access: diamond, 2015
Chaymaa Marouf   +6 more
openalex   +1 more source

A Survey of Software-Defined Networking: Past, Present, and Future of Programmable Networks

open access: yesIEEE Communications Surveys and Tutorials, 2014
B. A. A. Nunes   +4 more
semanticscholar   +1 more source

Integrated genomic and proteomic profiling reveals insights into chemoradiation resistance in cervical cancer

open access: yesMolecular Oncology, EarlyView.
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath   +13 more
wiley   +1 more source

Pendular See-Saw Nystagmus: A Rare Presenting Manifestation of Craniopharyngioma

open access: green, 2020
Rimesh Pal   +4 more
openalex   +2 more sources

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

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