Results 91 to 100 of about 918 (139)
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Veterinary Clinics of North America - Small Animal Practice, 2003
GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
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GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
exaly +3 more sources
Protein-Losing Enteropathies in Dogs
Veterinary Clinics of North America - Small Animal Practice, 2011Protein-losing enteropathy is common in dogs but rare in cats. In the vast majority of cases, it is associated with inflammatory bowel disease, intestinal lymphoma, or intestinal lymphangiectasia. The diagnosis is based on identification of hypoalbuminemia and ruling out urinary loss and liver failure.
Olivier Dossin, Rachel Lavoué
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2010
Protein-losing enteropathies are a syndrome caused by mucosal disruption or increased permeability or obstruction of lymphatic drainage. Documentation of excessive intestinal protein loss is the hallmark of the diagnosis. Treatment depends on the primary disease process and is usually a combination of dietary, medical, and/or surgical therapy.
Jonathan Goldstein
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Protein-losing enteropathies are a syndrome caused by mucosal disruption or increased permeability or obstruction of lymphatic drainage. Documentation of excessive intestinal protein loss is the hallmark of the diagnosis. Treatment depends on the primary disease process and is usually a combination of dietary, medical, and/or surgical therapy.
Jonathan Goldstein
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PROTEIN-LOSING ENTEROPATHIES IN MALIGNANCY
Annals of the New York Academy of Sciences, 1974S Broder, Warren Strober
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Pediatric Clinics of North America, 1962
SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
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SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
+5 more sources
Current Opinion in Gastroenterology, 2020
Purpose of review The present review offers its readers a practical overview of protein-losing enteropathy, particularly with regard to diagnostic and therapeutic approaches. The aim is to support clinicians in their daily practice with a practical tool to deal with protein-losing enteropathy ...
Elli L., Topa M., Rimondi A.
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Purpose of review The present review offers its readers a practical overview of protein-losing enteropathy, particularly with regard to diagnostic and therapeutic approaches. The aim is to support clinicians in their daily practice with a practical tool to deal with protein-losing enteropathy ...
Elli L., Topa M., Rimondi A.
openaire +2 more sources
Protein‐losing enteropathy in the Lundehund
Journal of Small Animal Practice, 1977ABSTRACTProtein‐losing enteropathy in the Lundehund, a possible hereditary disease, characterized clinically by intermittent diarrhoea, vomiting, wasting, ascites and oedema, is described. Serum examinations revealed low values of protein and calcium.Post‐mortem changes consisted of thickened duodenal and jejunal mucosa with blunt, short, anastomosing ...
K, Flesjå, T, Yri
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