Results 151 to 160 of about 3,502,764 (202)
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Pediatric Clinics of North America, 1962
SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
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SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
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Veterinary Clinics of North America: Small Animal Practice, 2003
GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
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GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
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Protein‐losing enteropathy in the Lundehund
Journal of Small Animal Practice, 1977ABSTRACTProtein‐losing enteropathy in the Lundehund, a possible hereditary disease, characterized clinically by intermittent diarrhoea, vomiting, wasting, ascites and oedema, is described. Serum examinations revealed low values of protein and calcium.Post‐mortem changes consisted of thickened duodenal and jejunal mucosa with blunt, short, anastomosing ...
K, Flesjå, T, Yri
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Protein-Losing Enteropathies in Dogs
Veterinary Clinics of North America: Small Animal Practice, 2011Protein-losing enteropathy is common in dogs but rare in cats. In the vast majority of cases, it is associated with inflammatory bowel disease, intestinal lymphoma, or intestinal lymphangiectasia. The diagnosis is based on identification of hypoalbuminemia and ruling out urinary loss and liver failure.
Olivier, Dossin, Rachel, Lavoué
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Cardiomyopathy with Protein-Losing Enteropathy
Chest, 1973A patient with cardiomyopathy, severe hypoproteinemia and lymphocytopenia is described. Increased catabolic rate of plasma albumin and excessive intestinal protein loss were documented. In spite of the combined lymphocytopenia and hypoimmunoglobulinemia, the patient was not predisposed to infections.
I, Oliver +4 more
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Protein-losing enteropathy and gastropathy
Current Treatment Options in Gastroenterology, 2001The diagnosis of protein-losing enteropathy (PLE) should be considered in all patients with hypoalbuminemia and edema without other known causes, and established by plasma alpha(1)-antitrypsin (alpha(1)-AT) clearance or nuclear studies. The therapy for PLE should focus principally on the treatment of the underlying disease after it has been identified.
Brian R., Landzberg, Mark B., Pochapin
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Protein‐Losing Enteropathy in a Dog
Journal of Small Animal Practice, 1969Abstract— —A protein‐losing enteropathy in a dog is described. There was a history of chronic gastrointestinal upset and examination revealed hypoproteinaemic oedema and ascites. Antemortem laboratory findings indicated a protein‐losing enteropathy and these, together with the results of clinical and necropsy examination, are described.
B R, Farrow, R, Penny
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Giardiasis with Protein-Losing Enteropathy
Journal of Pediatric Gastroenterology and Nutrition, 1985Generalized edema and ascites were the main presenting features of giardiasis in a 3-year-old boy. Hypoalbuminemia, jejunal villous atrophy, Giardia lamblia in the duodenal aspirate, and abnormal gastrointestinal protein loss were present before therapy with metronidazole.
D L, Sutton, K R, Kamath
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A CASE OF PROTEIN‐LOSING ENTEROPATHY
Australasian Annals of Medicine, 1960SummaryThe case described is of a woman, aged 61 years, presenting with diarrhoea, fever and œdema associated with hypoproteinæmia. Biopsy of the intestine showed acute non‐specific ileitis, and biopsy of the liver showed severe fatty degeneration. Laparotomy revealed some vascular engorgement of the subserosal vessels in the lower ileal area.
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