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Pathophysiology of pulmonary edema
Critical Care Nursing Quarterly, 1994Pulmonary edema is a frequent and common cause of death in patients in critical care settings. It is seen as a complication of myocardial infarcts, hypertension, pneumonia, smoke inhalation, and high-altitude pulmonary edema. Pulmonary edema occurs when there are alterations in Starling forces and capillary permeability, opposition to lymphatic flow in
A D, Angerio, P A, Kot
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Noncardiogenic pulmonary edema
Emergency Medicine Clinics of North America, 2003Pulmonary edema is differentiated into two categories--cardiogenic and noncardiogenic. Noncardiogenic pulmonary edema is due to changes in permeability of the pulmonary capillary membrane as a result of either a direct or an indirect pathologic process. It is a spectrum of illness ranging from the less severe form of ALI to the severe ARDS.
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International Journal of Gynecology & Obstetrics, 2002
The objective of this study is to investigate factors related to the outcome of pregnancies complicated by pulmonary edema. A retrospective search of the previous 10 years records for pregnant patients with pulmonary edema was performed at a tertiary medical center.
W C, Huang, C P, Chen
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The objective of this study is to investigate factors related to the outcome of pregnancies complicated by pulmonary edema. A retrospective search of the previous 10 years records for pregnant patients with pulmonary edema was performed at a tertiary medical center.
W C, Huang, C P, Chen
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Current Treatment Options in Cardiovascular Medicine, 1999
Patients with acute cardiogenic pulmonary edema require rapid assessment and therapy to prevent progression to respiratory failure and cardiovascular collapse. The goal of therapy is to decrease the pulmonary capillary wedge pressure by decreasing intravascular volume and shifting the blood volume into peripheral vascular beds.
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Patients with acute cardiogenic pulmonary edema require rapid assessment and therapy to prevent progression to respiratory failure and cardiovascular collapse. The goal of therapy is to decrease the pulmonary capillary wedge pressure by decreasing intravascular volume and shifting the blood volume into peripheral vascular beds.
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Pulmonary Edema II: Noncardiogenic Pulmonary Edema
DeckerMed Transitional Year Weekly Curriculum™, 2021Gerald W. Staton Jr +2 more
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Neurologic Clinics, 1984
The pathogenesis of nervous system-induced pulmonary edema remains incompletely understood. There are two major causes: elevated intravascular pressure and pulmonary capillary leak. Thus, both hemodynamic (cardiogenic) or nonhemodynamic (noncardiogenic) components exist.
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The pathogenesis of nervous system-induced pulmonary edema remains incompletely understood. There are two major causes: elevated intravascular pressure and pulmonary capillary leak. Thus, both hemodynamic (cardiogenic) or nonhemodynamic (noncardiogenic) components exist.
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