Results 291 to 300 of about 146,623 (321)
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Chest, 1982
This report presents a patient who developed fulminant pulmonary edema as a complication of an acute subarachnoid hemorrhage. Hemodynamic evaluation revealed low-normal pulmonary arteriolar resistances. Endobronchial fluid was freely suctioned from the patient over a two-day period and had a colloid osmotic pressure and protein content equal to the ...
Alan Fein, Eric C. Rackow
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This report presents a patient who developed fulminant pulmonary edema as a complication of an acute subarachnoid hemorrhage. Hemodynamic evaluation revealed low-normal pulmonary arteriolar resistances. Endobronchial fluid was freely suctioned from the patient over a two-day period and had a colloid osmotic pressure and protein content equal to the ...
Alan Fein, Eric C. Rackow
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Chest, 1974
The integrity of the normal endothelial barrier is responsible for two of the three major safety factors preventing pulmonary edema. This is why edema due to increased pressure is usually not as severe as edema due to increased permeability. Management ought to follow sound physiologic principles. These principles are to lower microvascular hydrostatic
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The integrity of the normal endothelial barrier is responsible for two of the three major safety factors preventing pulmonary edema. This is why edema due to increased pressure is usually not as severe as edema due to increased permeability. Management ought to follow sound physiologic principles. These principles are to lower microvascular hydrostatic
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Noncardiogenic pulmonary edema [PDF]
Emergency Medicine Clinics of North America, 2003 Pulmonary edema is differentiated into two categories--cardiogenic and noncardiogenic. Noncardiogenic pulmonary edema is due to changes in permeability of the pulmonary capillary membrane as a result of either a direct or an indirect pathologic process. It is a spectrum of illness ranging from the less severe form of ALI to the severe ARDS.
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Pulmonary Edema and Propoxyphene
JAMA: The Journal of the American Medical Association, 1971To the Editor.— The article "Pulmonary Edema Associated with Propoxyphene Intoxicition" ( 215 :259-262, 1971), described the phenomenon of acute pulmonary edema, congestion, and hemorrhage that is seen daily in all manners of death by the forensic pathologist. These deaths range from sudden death due to cardiac arrhythmias, to barbiturate overdose, to
Vincent J. M. DiMaio, Lawrence D. Henry
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Neurologic Clinics, 1984
The pathogenesis of nervous system-induced pulmonary edema remains incompletely understood. There are two major causes: elevated intravascular pressure and pulmonary capillary leak. Thus, both hemodynamic (cardiogenic) or nonhemodynamic (noncardiogenic) components exist.
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The pathogenesis of nervous system-induced pulmonary edema remains incompletely understood. There are two major causes: elevated intravascular pressure and pulmonary capillary leak. Thus, both hemodynamic (cardiogenic) or nonhemodynamic (noncardiogenic) components exist.
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Clinics in Chest Medicine, 1985
Neurogenic pulmonary edema is an anomaly because it cannot be categorized into either of the two major types of pulmonary edema. Both high-pressure and increased-permeability abnormalities may be involved in the pathogenesis of neurogenic pulmonary edema. Furthermore, the mechanisms responsible for these abnormalities appear quite complex.
Gene L. Colice, Gene L. Colice
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Neurogenic pulmonary edema is an anomaly because it cannot be categorized into either of the two major types of pulmonary edema. Both high-pressure and increased-permeability abnormalities may be involved in the pathogenesis of neurogenic pulmonary edema. Furthermore, the mechanisms responsible for these abnormalities appear quite complex.
Gene L. Colice, Gene L. Colice
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Acta Anaesthesiologica Scandinavica, 2007
Neurogenic pulmonary edema (NPE) is usually defined as an acute pulmonary edema occurring shortly after a central neurologic insult. It has been reported regularly for a long time in numerous and various injuries of the central nervous system in both adults and children, but remains poorly understood because of the complexity of its pathophysiologic ...
Antoine Baumann +3 more
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Neurogenic pulmonary edema (NPE) is usually defined as an acute pulmonary edema occurring shortly after a central neurologic insult. It has been reported regularly for a long time in numerous and various injuries of the central nervous system in both adults and children, but remains poorly understood because of the complexity of its pathophysiologic ...
Antoine Baumann +3 more
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The American Journal of Medicine, 1978
Pulmonary edema fluid analyses and hemodynamic evaluations were performed in two uremic patients with acute pulmonary edema. The colloid osmotic pressure of the pulmonary edema fluid ranged from 57 per cent to 93 per cent that of the serum. Although cardiac function was normal in both patients, the serum colloid osmotic pressure--pulmonary artery wedge
Eric C. Rackow +3 more
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Pulmonary edema fluid analyses and hemodynamic evaluations were performed in two uremic patients with acute pulmonary edema. The colloid osmotic pressure of the pulmonary edema fluid ranged from 57 per cent to 93 per cent that of the serum. Although cardiac function was normal in both patients, the serum colloid osmotic pressure--pulmonary artery wedge
Eric C. Rackow +3 more
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Chest, 1973
A 21-year-old man was in a coma and had pulmonary edema after ingesting 80 mg of methadone orally. Prompt use of naloxone hydrochloride reversed the respiratory depression. The diagnosis and treatment of methadone-induced pulmonay edema is reviewed.
Robert W. Enquist, Allan L. Goldman
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A 21-year-old man was in a coma and had pulmonary edema after ingesting 80 mg of methadone orally. Prompt use of naloxone hydrochloride reversed the respiratory depression. The diagnosis and treatment of methadone-induced pulmonay edema is reviewed.
Robert W. Enquist, Allan L. Goldman
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Postobstruction Pulmonary Edema
Chest, 1989Several occurrences of pulmonary edema following relief of acute upper airway obstruction have been reported. The edema is associated with normal cardiac filling pressures and responds promptly to conservative therapy. Its origin may be attributed to the cardiopulmonary effects of the vigorous inspiratory effort that the spontaneously breathing patient
Lewis A. Coveler +2 more
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