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Pathobiology of pulmonary fibrosis
American Journal of Physiology-Lung Cellular and Molecular Physiology, 1990Pulmonary fibrosis is characterized by an increase in lung matrix and alterations in the numbers and spatial relationships of lung parenchymal cells. The increase in matrix results from a proliferation and “activation” of fibroblasts (FB) with increased production and deposition of matrix macromolecules at sites of lung injury.
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Macrophages and Pulmonary Fibrosis
Current Molecular MedicineMost chronic respiratory diseases often lead to the clinical manifestation of pulmonary fibrosis. Inflammation and immune disorders are widely recognized as primary contributors to the onset of pulmonary fibrosis. Given that macrophages are predominantly responsible for inflammation and immune disorders, in this review, we first focused on the role ...
Shengjun, Chen +2 more
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Virchows Archiv A Pathological Anatomy and Histology, 1977
J C, Belton, N, Crise
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J C, Belton, N, Crise
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Pulmonary Complications in Cancer Patients
Ca-A Cancer Journal for Clinicians, 1996Robert J Kaner +2 more
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Prednisone, Azathioprine, and N-Acetylcysteine for Pulmonary Fibrosis
New England Journal of Medicine, 2012Kevin J Anstrom +2 more
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Surgical Management of Pulmonary Metastases
Ca-A Cancer Journal for Clinicians, 1963E W Wilkins
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