Results 191 to 200 of about 605,155 (296)

Clinical Predictors and Recurrence Characteristics Following Radiotherapy for Primary Central Nervous System Lymphoma: A Retrospective Cohort Study. [PDF]

Cancers (Basel)
Bigge JC, Bendrich S, Treiber H, Aydilek E, Brökers N, Wulf GG, Zwerenz CM, Anczykowski MZ, Donath S, El Shafie RA, von Diest LA, Oelmann JT, Schirmer MA, Dröge LH, Leu M, Chapuy B, Rieken S, Guhlich M.   +17 more
europepmc   +1 more source

Natulan (Procarbazine) combined with radiotherapy in management of inoperable malignant melanoma. [PDF]

, 1965
Geoffrey Falkson, Peter Villiers, H. C. Falkson, T Fichardt   +3 more
openalex   +1 more source

The critical role of DNA damage‐inducible transcript 4 (DDIT4) in stemness character of leukemia cells and leukemia initiation

Molecular Oncology, EarlyView.
Stemness properties, including quiescence, self‐renewal, and chemoresistance, are closely associated with leukemia relapse. Here, we demonstrate that DNA damage‐inducible transcript 4 (DDIT4) is induced in the hypoxic bone marrow niche and is essential for maintaining the stemness of AML1‐ETO9a leukemia cells.
Yishuang Li, Zhijie Cao, Haiyan Xing, Zhenya Xue, Wenbing Liu, Jiayuan Chen, Yihan Mei, Runxia Gu, Hui Wei, Shaowei Qiu, Min Wang, Qing Rao, Jianxiang Wang   +12 more
wiley   +1 more source

Gut microbiota and radiation-induced injury: mechanistic insights and microbial therapies. [PDF]

Gut Microbes
Li L, Yang Z, Yi Y, Song Y, Zhang W.
europepmc   +1 more source

Radiotherapy for Carcinoma of the Lung [PDF]

, 1965
Ulla Beling, Jerzy Einhorn
openalex   +1 more source

Unveiling unique protein and phosphorylation signatures in lung adenocarcinomas with and without ALK, EGFR, and KRAS genetic alterations

Molecular Oncology, EarlyView.
Proteomic and phosphoproteomic analyses were performed on lung adenocarcinoma (LUAD) tumors with EGFR, KRAS, or EML4–ALK alterations and wild‐type cases. Distinct protein expression and phosphorylation patterns were identified, especially in EGFR‐mutated tumors. Key altered pathways included vesicle transport and RNA splicing.
Fanni Bugyi, Mirjam Balbisi, Simon Sugár, Lóránd Váncza, Eszter Regős, Ilona Kovalszky, Ibolya Laczó, Tünde Harkó, Gábor Kecskeméti, Zoltán Szabó, Judit Moldvay, László Drahos, Lilla Turiák   +12 more
wiley   +1 more source

Histologic effects of endolymphatic radiotherapy [PDF]

, 1969
Reginald S. A. Lord, J. B. Kinmonth
openalex   +1 more source

Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs

Molecular Oncology, EarlyView.
In HGSOC with normal KRAS expression, high PTP4A3 expression regulates autophagy activation. Conversely, in HGSOC with high KRAS expression, KRAS dictates autophagy control, and PTP4A3 is not required. When high PTP4A3 expression is inhibited, HGSOC cells are preferentially sensitised towards DNA‐damaging agents.
Ana López‐Garza, David James, Emma Creagh, James T. Murray   +3 more
wiley   +1 more source

De-escalation of radiotherapy in the treatment of human papillomavirus-associated oropharyngeal cancer. [PDF]

Cochrane Database Syst Rev
Lawless AK, Duruchukwu E, Bergamin S, Jayamanne DT, Back M, Lee A.   +5 more
europepmc   +1 more source

Roentgen Stand for Field Positioning in High-Energy Radiotherapy [PDF]

, 1968
Bo Jung, Börje Larsson, Bengt Rosengren, K. Stahl, Wilhelm Wretlind   +4 more
openalex   +1 more source