Results 111 to 120 of about 20,153 (290)

Compressions of Resolvents and Maximal Radius of Regularity

open access: yes, 1999
Suppose that $\lambda - T$ is left-invertible in $L(H)$ for all $\lambda \in \Omega$, where $\Omega$ is an open subset of the complex plane. Then an operator-valued function $L(\lambda)$ is a left resolvent of $T$ in $\Omega$ if and only if $T$ has an ...
Badea, C., Mbekhta, M.
core  

Adenosine‐to‐inosine editing of miR‐200b‐3p is associated with the progression of high‐grade serous ovarian cancer

open access: yesMolecular Oncology, EarlyView.
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira   +14 more
wiley   +1 more source

Trace expansions for elliptic cone operators with stationary domains

open access: yes, 2009
We analyze the behavior of the trace of the resolvent of an elliptic cone differential operator as the spectral parameter tends to infinity. The resolvent splits into two components, one associated with the minimal extension of the operator, and another,
Gil, Juan B.   +2 more
core   +1 more source

A Representation Formula for Strongly Continuous Resolvent Families

open access: yesJournal of Integral Equations and Applications, 1997
The resolvent of the linear Volterra equation \[ u(t)=f(t)+\int_0^ta(t-s)Au(s)ds \] in a Banach space with a closed operator \(A\) is related to \(a\) by a Laplace transform. This relation and a result on the inverse Laplace transform by \textit{B. Hennig} and \textit{F. Neubrander} [Appl. Anal. 49, No.
openaire   +2 more sources

Integrated genomic and proteomic profiling reveals insights into chemoradiation resistance in cervical cancer

open access: yesMolecular Oncology, EarlyView.
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath   +13 more
wiley   +1 more source

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

A Study of Positive Solutions for Semilinear Fractional Measure Driven Functional Differential Equations in Banach Spaces

open access: yesMathematics
In this paper, we deal with the delayed measure differential equations with nonlocal conditions via measure of noncompactness in ordered Banach spaces. Combining (β,γk)-resolvent family, regulated functions and fixed point theorem with respect to convex ...
Jing Zhang, Haide Gou
doaj   +1 more source

The 𝑒-positive mild solutions for fractional impulsive evolution equations with 1 < 𝛼 < 2

open access: yesCQD Revista Eletrônica Paulista de Matemática, 2022
In this paper, we investigate the existence of global 𝑒-positive mild solutions for impulsive evolution fractional differential equation in the Caputo sense of order 1 < 𝛼 < 2.
Jorge Féres Junior   +2 more
doaj  

Emerging role of ARHGAP29 in melanoma cell phenotype switching

open access: yesMolecular Oncology, EarlyView.
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster   +3 more
wiley   +1 more source

Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine

open access: yesMolecular Oncology, EarlyView.
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu   +18 more
wiley   +1 more source

Home - About - Disclaimer - Privacy