Results 131 to 140 of about 19,341,281 (268)

Respect

open access: yesJournal of Investigative Surgery, 2005
openaire   +2 more sources

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

open access: yesMolecular Oncology, EarlyView.
This study indicates that Merkel cell carcinoma (MCC) does not originate from Merkel cells, and identifies gene, protein & cellular expression of immune‐linked and neuroendocrine markers in primary and metastatic Merkel cell carcinoma (MCC) tumor samples, linked to Merkel cell polyomavirus (MCPyV) status, with enrichment of B‐cell and other immune cell
Richie Jeremian   +10 more
wiley   +1 more source

Grooming Behavior in Diplura (Insecta: Apterygota)

open access: yesPsyche: A Journal of Entomology, 1978
Barry D. Valentine, Michael J. Glorioso
doaj   +1 more source

Major Determinants of Female Child Labour in Urban Multan (Punjab-Pakistan)

open access: yesLahore Journal of Economics
In recent years, the sensitive issue of child labour has received world-wide attention and has become the focus of serious discussion in developing as well as developed countries.
Karamat Ali, Abdul Hamid
doaj  

Integrated genomic and proteomic profiling reveals insights into chemoradiation resistance in cervical cancer

open access: yesMolecular Oncology, EarlyView.
A comprehensive genomic and proteomic analysis of cervical cancer revealed STK11 and STX3 as a potential biomarkers of chemoradiation resistance. Our study demonstrated EGFR as a therapeutic target, paving the way for precision strategies to overcome treatment failure and the DNA repair pathway as a critical mechanism of resistance.
Janani Sambath   +13 more
wiley   +1 more source

Respect Defended

open access: yesTrends in Cognitive Sciences, 2019
Engelmann, Jan M., Tomasello, Michael
openaire   +3 more sources

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

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