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The Adult Respiratory Distress Syndrome
Mayo Clinic Proceedings, 1982Acute lung injury from a variety of causes may result in capillary leakage and the nonhydrostatic edema of the adult respiratory distress syndrome. The mechanism of development of this syndrome is not yet fully understood. There is increasing hypoxemia from disturbance of normal ventilation-perfusion relationships in the lung and progressive loss of ...
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Adult respiratory distress syndrome in the tropics
Clinics in Chest Medicine, 2002Today ARDS is more frequently recognized and managed in tropical countries, although published data from most locations is meager. The spectrum of disorders causing ARDS in tropical countries includes virtually all conditions encountered in the West. Additionally, tropical infections and other disorders are seen far more commonly.
Surinder K, Jindal +2 more
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Adult Respiratory Distress Syndrome
Southern Medical Journal, 1978Treatment of the adult respiratory distress syndrome requires an understanding of the current concepts of the pathogenesis of this syndrome. The clinical features and pathophysiology are briefly discussed. Differential diagnosis requires the exclusion of pulmonary infection and left heart failure.
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Managing respiratory distress syndrome in adults
Postgraduate Medicine, 1976Respiratory distress syndrome in adults may be produced by any of a number of causes resulting in similar pathophysiologic changes and having identical therapeutic implications. The most important factors in treatment are early recognition and early institution of therapy.
J F, Tomashefski, V, Mahajan
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Acute respiratory distress syndrome in adults
Hospital Medicine, 2000The acute respiratory distress syndrome (ARDS) can arise from a range of predisposing insults. Mortality rates are high for patients with ARDS and survivors require extended and expensive intensive care treatment. This article presents evidence that implicates the production of toxic and damaging reactive oxygen species in the pathogenesis of ARDS.
G J, Quinlan, T W, Evans
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Adult respiratory distress syndrome — an update
Histopathology, 1999In adults, acute lung injury or adult respiratory distress syndrome (ARDS) may complicate a wide range of serious medical and surgical conditions, only some of which involve direct pulmonary insult. The characteristic histological feature of ARDS is an intense inflammatory process in the lungs, which may progress to fibrosis. The earliest physiological
P S, Hasleton, T E, Roberts
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Adult Respiratory Distress Syndrome in Children
Pediatric Clinics of North America, 1980Respiratory distress syndrome is the clinical manifestation of injury to the terminal alveolar-capillary unit, and may result from a variety of nonpulmonary insults including shock, sepsis, and trauma. The clinical characteristics, pathophysiology, and treatment of respiratory distress syndrome in children are reviewed.
P R, Holbrook +3 more
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Treatment of Adult Respiratory Distress Syndrome
Drug Intelligence & Clinical Pharmacy, 1988Adult respiratory distress syndrome (ARDS) culminates in inadequate oxygen delivery to the tissues. There are numerous inciting factors for this syndrome. Several therapies including the prophylactic use of antibiotics and steroids are controversial; even mechanical ventilatory support has controversial elements.
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Adult Respiratory Distress Syndrome in Babesiosis
Chest, 1984A patient is described who contracted transfusion-induced babesiosis, and later developed acute respiratory distress syndrome (ARDS) as a fatal complication. ARDS has been reported in patients with Plasmodium falciparum malaria, but to our knowledge has not been observed as a complication of babesiosis.
S, Gordon +5 more
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Adult Respiratory Distress Syndrome in Children
Pediatric Clinics of North America, 1994In spite of modern technological advances, ARDS continues to be an important cause of morbidity and mortality from a diverse group of disorders such as sepsis, trauma, and aspiration. ARDS represents a target organ injury resulting from activation of the host's inflammatory cells and uncontrolled liberation of inflammatory mediators. In most instances,
A P, Sarnaik, M, Lieh-Lai
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