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A case of pure retrograde amnesia following mild head injury is reported. Neuropsychological, psychodynamic and statistical approaches are employed in an attempt to disentangle the clinical picture presented by the patient.
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THE ÆTIOLOGY OF RETROGRADE AMNESIA
The Lancet, 1971Abstract To date, hypotheses of the aetiology of retrograde amnesia have focused on anatomical effects of traumatic lesions and on the presumed existence of short-term memory. The invariable association of retrograde amnesia with post-traumatic amnesia has been ignored as a clue to the origin of retrograde amnesia.
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Isolated Prolonged Retrograde Amnesia
European Neurology, 2008We describe a patient who had isolated retrograde amnesia of 1-year duration without anterograde amnesia after recovery from encephalitis. Single photon emission computed tomography using 123I-IMP revealed the left temporal lobe abnormality. We postulate that the learning ability for new information and recalling ability for old information involve ...
Y, Yoneda +3 more
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Retrograde Amnesia in Petit Mal
Archives of General Psychiatry, 1962In our former research on the alterations of consciousness in petit mal (Jus and Jus, 1960) we were interested in different kinds of anterograde amnesia in petit mal and also devoted some attention to the problem of amnesia. In the present study we have made a more precise analysis of retrograde amnesia in petit mal.
A, JUS, K, JUS
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1967
Publisher Summary The retrograde amnesia (RA) phenomenon occurs in both animals and man and overwhelming evidence exists showing that a single electroconvulsive shock (ECS) treatment given after acquisition produces RA. Because ECS constitutes such a reliable RA treatment, drugs can be studied for their ability to duplicate the effects of this ...
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Publisher Summary The retrograde amnesia (RA) phenomenon occurs in both animals and man and overwhelming evidence exists showing that a single electroconvulsive shock (ECS) treatment given after acquisition produces RA. Because ECS constitutes such a reliable RA treatment, drugs can be studied for their ability to duplicate the effects of this ...
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Caudate stimulation and retrograde amnesia: Amnesia threshold and gradient
Behavioral Biology, 1972This experiment determined the threshold stimulation intensity needed for production of retrograde amnesia with 60 Hz caudate stimulation. With the stimulation parameters used, the necessary intensity is between 1 and 1.5 mA. This stimulation is an effective amnesic agent when administered 15 min, but not 60 min, after training.
P E, Gold, R A, King
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Aphasiology, 1995
Abstract A case of severe retrograde and less severe anterograde amnesia with initial multimodal agnosia is described as an outcome of traumatic brain damage. Anatomical and metabolic studies document right frontal and left occipital lesions with sparing of medial temporal cortex.
Jason W. Brown, Karen L. Chobor
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Abstract A case of severe retrograde and less severe anterograde amnesia with initial multimodal agnosia is described as an outcome of traumatic brain damage. Anatomical and metabolic studies document right frontal and left occipital lesions with sparing of medial temporal cortex.
Jason W. Brown, Karen L. Chobor
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Retrograde amnesia during transient global amnesia
Neurocase, 1996Abstract Two patients who met Hodges' clinical criteria for transient global amnesia (TGA) were given anterograde and retrograde memory tests during and after the attack. A SPECT scan was performed during TGA in one case, showing a reduced blood flow confined to the bilateral medial temporal lobes, which resolved on the next day.
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Retrograde Amnesia in Free Recall
Science, 1969Supervention of high-priority events in a series of events constituting a free-recall task interferes with postexposure processing of mnemonic information about immediately preceding events, with the result that recall of these preceding events is impaired. Recall of immediately following events is not affected.
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The syndrome of pure retrograde amnesia
Neurocase, 1998Three cases of pure retrograde amnesia (PRA) are reported. The sudden onset of PRA occurred after minor head trauma in two cases (CDA and AF) and after emotional stress in one case (GC). No structural brain damage was apparent in any of the three patients. Evidence of temporal gradient was found.
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