Results 151 to 160 of about 365,073 (343)
Hot Hole-Induced Device Degradation By Drain Junction Reverse Current [PDF]
Kwangsoo Kim +3 more
openalex +1 more source
UiO‐66(Zr) metal–organic frameworks are chemically stable, biocompatible, and highly tunable nanomaterials. Their modular structure enables controlled drug delivery, multimodal bioimaging, and light‐activated photodynamic therapy, supporting integrated diagnostic and therapeutic (theranostic) applications in cancer and biomedical research.
Veronika Huntošová +2 more
wiley +1 more source
Natural products target the aging kidney in diabetic nephropathy by restoring the AMPK–SIRT1–Nrf2 axis, reducing oxidative stress, inflammation, fibrosis, and cellular senescence while enhancing mitochondrial biogenesis and antioxidant defenses.
Sherif Hamidu +8 more
wiley +1 more source
The influence of frequency of alternating or infrequently reversed current on electrolytic corrosion
Burton McCollum, G. H. Ahlborn
openalex +2 more sources
Aging Is a Key Driver for Adult Acute Myeloid Leukemia
Acute myeloid leukemia (AML) is a classical age‐related hematologic malignancy, and a key driver of AML is aging, which profoundly regulates intrinsic factors such as genomic instability, epigenetic reprogramming, and metabolic dysregulation, and alters bone marrow microenvironment.
Rong Yin, Haojian Zhang
wiley +1 more source
Ultrafast optical control of ferroelectricity using short, intense light pulses enables precise manipulation of ferroelectric materials, offering potential breakthroughs in nonvolatile memory technologies.
Ali Kazempour +6 more
doaj +1 more source
Reverse Remodeling and Current Medical Therapy in Heart Failure with Reduced Ejection Fraction
Hisataka Maki, Norihiko Takeda
openalex +2 more sources
Mutant NPM1 in Acute Myeloid Leukemia Initiation and Maintenance
NPM1 mutations drive acute myeloid leukemia by acting as neomorphic transcriptional regulators that cooperate with Menin–MLL and XPO1 to sustain HOX/MEIS1 expression and block differentiation. Targeting these mutant‐specific transcriptional dependencies provides a rational therapeutic strategy for NPM1‐mutated AML.
Yanan Jiang +3 more
wiley +1 more source

