Results 71 to 80 of about 1,499,548 (192)
FEBS Letters, EarlyView.Vacuolar H+‐ATPase (V‐ATPase) is a multisubunit rotary proton pump that acidifies intracellular compartments. Using elastic network models, we identified conserved hinge residues that govern its large‐scale functional motions. We show that intrinsically encoded ‘soft modes’ drive critical functional transitions, including rotary catalysis and ...
Diksha Parwana +3 more
wiley +1 more source
Ehsan200/reviewer-recommender: 1.0.0
<h1>Release 1.0.0</h1> <p>Implemented a reviewer recommender simulator that uses different algorithms (<code>RevFinder</code>, <code>ChRev</code>, <code>TurnoverRec</code>, and <code>Sofia< ...
Ehsan Movaffagh, SAREGH
core +1 more source
Mid-Western Educational Research Association Reviewers for 2005-2006 [PDF]
, 2007 List of ...
Rodgers, Adrian, Jenkins, Deborah Bainer
core
The role of miR‐335‐5p in the redifferentiation of BRAF p.V600E thyroid cancers
Molecular Oncology, EarlyView.The BRAF p.V600E mutation promotes thyroid cancer dedifferentiation and radioiodine resistance. Using a network approach, we identified miR‐335‐5p as a key regulator of BRAF‐mutated thyroid tumors. Restoring miR‐335‐5p increased thyroid‐specific gene expression and iodine uptake in cells and organoids.
Valeria Pecce +11 more
wiley +1 more source
Cover Volume 10(4) October 2021
, 2021 The file content of: editorial list, reviewer list, table of ...
JAcPS, JAcPS
core
KDM7A and KDM1A inhibition suppresses tumour promoting pathways in prostate cancer
Molecular Oncology, EarlyView.Treatment resistance is a major challenge for patients with advanced prostate cancer. This study examined an alternative approach to target the major prostate cancer‐promoting pathway by targeting epigenetic factors, whose levels are higher in tumours.
Jennie N Jeyapalan +16 more
wiley +1 more source
Cover Volume 10(1) January 2021
, 2021 The file content of: editorial list, reviewer list, table of ...
JAcPS, Editorial
core
Molecular Oncology, EarlyView.This study shows that lung adenocarcinomas exploit developmental branching morphogenesis to acquire a therapy resistant basal‐like tumour cell state. This process was found to be regulated by combined TP53 loss‐of‐function and type‐I interferon signalling, identifying a novel axis for biomarker and therapeutic target discovery.
Kamila J Bienkowska +13 more
wiley +1 more source