Results 171 to 180 of about 49,127 (197)

Spontaneous Reye's-like syndrome in BALB/cByJ mice.

Laboratory investigation; a journal of technical methods and pathology, 1984
In five spontaneous outbreaks, sixty-four BALB/cByJ mice developed Reye's-like syndrome 4 to 33 days after introduction into mouse rooms known to harbor a variety of indigenous murine viruses. The clinical course lasted 24 hours and consisted of progressively deteriorating consciousness and hyperventilation, usually leading to death.
D G, Brownstein, E A, Johnson, A L, Smith   +2 more
openaire   +1 more source

Recurrent Reye-like syndrome: possible association with Krebs cycle abnormality.

Israel journal of medical sciences, 1984
During a 7-year period, a 15-year-old boy experienced recurrent attacks that clinically and pathologically resembled Reye's syndrome. The attacks were precipitated by prolonged exercise, fasting or infections. An extensive investigation failed to identify a toxic cause or specific metabolic disorder.
E, Granot, R J, Deckelbaum, R, Judd, A E, Slonim, A, Gutman   +4 more
openaire   +1 more source

Reye-like syndrome associated with valproic acid therapy

The Journal of Pediatrics, 1979
N, Gerber, R G, Dickinson, R C, Harland, R K, Lynn, L D, Houghton, J I, Antonias, J C, Schimschock   +6 more
openaire   +2 more sources

[3-hydroxy-3-methylglutaric aciduria and recurrent Reye-like syndrome].

Revista de neurologia, 1998
3-hydroxy-3-methylglutaryl-coenzyme A lyase deficiency (HMG-CoA lyase) is an inborn error of ketogenesis and Leucine catabolism. HMG-CoA lyase catalyses the final step in leucine degradation, converting HMG-CoA to acetyl-CoA and acetoacetic acid. Clinical manifestations include hepatomegaly, lethargy or coma and apnoea.
J, Eirís, A, Ribes, R, Fernández-Prieto, J, Rodríguez-García, S, Rodríguez-Segade, M, Castro-Gago   +5 more
openaire   +1 more source

Fatal reye‐like syndrome associated with valproic acid

Annals of Neurology, 1980
Richard S. K. Young, Ira Bergman, David L. Gang, Edward P. Richardson   +3 more
openaire   +1 more source

Hereditary fructose intolerance presenting as Reye's-like syndrome: report of one case.

Acta paediatrica Taiwanica = Taiwan er ke yi xue hui za zhi, 2000
Hereditary fructose intolerance (HFI) is an autosomal recessive disease caused by catalytic deficiency of aldolase B (fructose-1, 6-bisphosphate aldolase). Herein we report on a case of hereditary fructose intolerance with initial presentation of episodic unconsciousness, seizure, hypoglycemia, hepatomegaly, and abnormal liver function since the ...
T Y, Yang, H L, Chen, Y H, Ni, W L, Hwu, M H, Chang   +4 more
openaire   +1 more source

Fructose-1,6-Diphosphatase Deficiency Presenting as a Reye-like Syndrome

Journal of Pediatric Endocrinology and Metabolism, 1987
Stephanos Mantagos, Constantine Frimas, Basil Thanopoulos, Nicholas G. Beratis   +3 more
openaire   +1 more source

Reye-like syndrome induced by valproic acid

Journal of Hepatology, 1986
openaire   +1 more source

Infantile disease with microvesicular fatty infiltration of viscera spontaneously occurring in the C3H-H-2° strain of mouse with similarities to Reye's syndrome

Laboratory Animals, 1988
J Hayakawa, Akitaka Nonomura
exaly  

Animal model of margosa oil ingestion with Reye-like syndrome. Pathogenesis of microvesicular fatty liver

Journal of Pathology, 1989
R Sinniah, D Sinniah
exaly