Results 1 to 10 of about 82,393 (306)
Nature, 1948 IN his address to the Seventh Congress of Biological Chemistry in Liege on October 3–6, 1946, Dr. H. D. Kay summarized some experiments on feeding pigs with yeast, carried out during the War at the National Institute for Research in Dairying in Reading1.
E. Hoff‐Jørgensen
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An Overview of Rickets in Children
Kidney International Reports, 2020 Rickets is a common bone disease worldwide that is associated with disturbances in calcium and phosphate homeostasis and can lead to short stature and joint deformities.
Rahul Chanchlani +7 more
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Nature Reviews Disease Primers, 2017 Rickets is a bone disease associated with abnormal serum calcium and phosphate levels. The clinical presentation is heterogeneous and depends on the age of onset and pathogenesis but includes bowing deformities of the legs, short stature and widening of joints. The disorder can be caused by nutritional deficiencies or genetic defects.
Thomas O Carpenter +2 more
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Management of rickets: the new horizons for the pediatrician [PDF]
Journal of Health, Population and NutritionRickets is a major public health concern globally. It results from impaired mineralization of the growing bone at its growth plate associated with abnormal calcium and phosphate metabolism.
Rummana Tazia Tonny +4 more
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Nutritional rickets and its associated factors among under-five children in Assela referral and teaching hospital, Ethiopia: a hospital based cross-sectional study design [PDF]
BMC PediatricsBackground Nutritional rickets is a preventable skeletal disorder caused by deficiencies in vitamin D, calcium, or phosphate, leading to softening and weakening of bones.
Helen Abebe +8 more
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Endokrynologia Polska, 2021 Hypophosphataemic rickets (HR) is a genetic disorder causing defects in the renal handling of phosphorus, resulting in rickets. HR can be classified into two groups.
Nahid Tavana +3 more
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A practical approach to diagnose and treat rickets [PDF]
Ķazaķstannyṇ Klinikalyķ Medicinasy, 2021 Rickets is a disease of growing bone, before fusion of epiphyses. There is defective mineralization of cartilage matrix in the zone of provisional calcification caused either by nutritional vitamin D deficiency and/or low calcium intake or by non ...
Aditi Jaiman +3 more
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Prevention of the disrupted enamel phenotype in Slc4a4-null mice using explant organ culture maintained in a living host kidney capsule. [PDF]
, 2014 Slc4a4-null mice are a model of proximal renal tubular acidosis (pRTA). Slc4a4 encodes the electrogenic sodium base transporter NBCe1 that is involved in transcellular base transport and pH regulation during amelogenesis.
Kurtz, Ira, Paine, Michael L, Wen, Xin
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